Diabetes Insipidus: Difference between revisions
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In the case of diabetes insipidus there is an increase in urine production, but without the sweet taste. | In the case of diabetes insipidus there is an increase in urine production, but without the sweet taste. | ||
Johann Peter Frank is credited with first making the distinction between diabetes mellitus and diabetes insipidus in 1794<ref>Fitz R. A Case of Diabetes Insipidus ''Arch Intern Med'' (1914) XIV(5): 706-721</ref>. The two main symptoms as excessive thirst and urination. | Johann Peter Frank is credited with first making the distinction between diabetes mellitus and diabetes insipidus (DI)in 1794<ref>Fitz R. A Case of Diabetes Insipidus ''Arch Intern Med'' (1914) XIV(5): 706-721</ref>. The two main symptoms as '''excessive thirst''' and '''excessive''' '''urination'''. | ||
Since then, DI has been attributed to a dysfunction in the production or action of the hormone vasopressin (AVP), affecting the individual's kidneys ability to retain water. The human body is made up of around 60-70% water, with the brain needing around 70-80% water. The blood requires around 50% water in the circulation to safely balance salts in our body. Therefore, in sever cases, it can be dangerous to the individual if DI is not identified and treated correctly to prevent too much, or too little hydration. | |||
== Clinically Relevant Anatomy == | == Clinically Relevant Anatomy == | ||
Revision as of 12:16, 31 October 2020
Top Contributors - Rhiannon Clement, Rucha Gadgil and Shaimaa Eldib
Introduction[edit | edit source]
Diabetes is a greek work meaning "siphon", and inspidus is the latin for "no taste".
The term for diabetes was coined for patients who pass of more fluid than they consume (therefore siphon) [1]. The urine was tested for sweetness. In the case of sweet urine the condition was names diabetes mellitus (latin for honey), and is the result of excess sugar in the urine and blood. In history this would be determined by examining the colour, sediment, odour and even the taste of the urine[1].
In the case of diabetes insipidus there is an increase in urine production, but without the sweet taste.
Johann Peter Frank is credited with first making the distinction between diabetes mellitus and diabetes insipidus (DI)in 1794[2]. The two main symptoms as excessive thirst and excessive urination.
Since then, DI has been attributed to a dysfunction in the production or action of the hormone vasopressin (AVP), affecting the individual's kidneys ability to retain water. The human body is made up of around 60-70% water, with the brain needing around 70-80% water. The blood requires around 50% water in the circulation to safely balance salts in our body. Therefore, in sever cases, it can be dangerous to the individual if DI is not identified and treated correctly to prevent too much, or too little hydration.
Clinically Relevant Anatomy[edit | edit source]
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Mechanism of Injury / Pathological Process[edit | edit source]
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Clinical Presentation[edit | edit source]
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Diagnostic Procedures[edit | edit source]
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Outcome Measures[edit | edit source]
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Management / Interventions[edit | edit source]
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Differential Diagnosis[edit | edit source]
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Resources[edit | edit source]
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