Anterior Ankle Impingement Syndrome: Difference between revisions

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<div class="noeditbox">Welcome to [[Vrije Universiteit Brussel Evidence-based Practice Project|Vrije Universiteit Brussel's Evidence-based Practice project]]. This space was created by and for the students in the Rehabilitation Sciences and Physiotherapy program of the Vrije Universiteit Brussel, Brussels, Belgium. Please do not edit unless you are involved in this project, but please come back in the near future to check out new information!!</div> <div class="editorbox">
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== Search Strategy  ==
== Definition/Description:                                                                                                                                                      ==


1. SEARCH STRATEGY ARTICLES
The anterior impingement syndrome of the ankle is a strangulation that can be caused by soft tissue, like the joint capsule or scar tissue, and hard tissue which refers to bone tissue. It’s location is the anterior side of the ankle in the talocrural joint. Due to repeated micro trauma’s little the ankle, the body will respond to this by building extra bone tissue called “osteophytes”. There are 3 types of osteophytes:     


<br>Keywords: <br>• Coccyx fracture ( 55 results and 3 free full texts): Most successful search<br>• Coccygectomy (85 results and 16 free full texts)<br>• Coccygeoplasty (1 free full text). <br>• Coccyx fracture AND physiotherapy (1 result)
*Traction spur.
*Inflammatory spur.
*Osteophyte or chondro-osteophyte.


<br>Search engines : Pubmed / Web of knowledge<br>It is also very useful to check the references of each usable article.
The one occurring here is a '''chondro-osteophyte'''.It is a defense mechanism of the body that prevents further injury, but reduces mobility and can lead to pain due to an impingement. There may potentially be swelling of the capsule and connective tissue.<ref name=":0" /> The relative contributions of the osseous and soft-tissue abnormalities are variable, but whatever component is dominant there physical impingement and painful limitation of ankle movement.<ref>Cerezal L, Abascal F, Canga A, Pereda T, García-Valtuille R, Pérez-Carro L, Cruz A. MR imaging of ankle impingement syndromes. American Journal of Roentgenology. 2003 Aug;181(2):551-9.</ref>     
== Clinically Relevant Anatomy:  ==


<br>2. SEARCH STRATEGY BOOKS
*The joint capsule is ventral and dorsal minimally present, it is reinforced with ligaments the lateral side. At the the tendons of the lower leg muscles, at the back is the tendon of the medial flexor . These muscles prevent the parts of the joint capsule from getting trapped between bones.[[File:Ank1.jpg|right|frameless|231x231px]]
*Foot free: In dorsiflexion the distal end of the talus moves lateral and the plantar side of the talus rotates to lateral. In plantar flexion the distal end of the talus moves medial, the plantar side of the talus rotates to medial.
*&nbsp;Foot stabilized on the floor: dorsal flexion provokes of the tibia with the distal end of the tibia moves to medial. Plantar flexion provokes an exorotation of the tibia with the distal end of the tibia moves to lateral Stabilityin the talocrural joint is the weakest in neutral position, this is because in this position the ligaments that surround the ankle are less tensed than in dorsal flexion or plantar flexion and there is less contact between joint facets. Plantar flexion has more stability, there is more contact between the joint facets and the tibionavicular part as well as the tibiotalar anterior part of the ligament deltoideum. The greatest stability occurs in dorsiflexion, the large front part of the trochlea tali now comes in touch with the small, narrow back part of joint socket, when this happens, the tibia and the fibula widen a bit and keep the talus closely bound helped with the strong tibiofibular ligaments.<ref name=":0">Cerezal L, Abascal F, Canga A, Pereda T, García-Valtuille R, Pérez-Carro L, Cruz A. MR imaging of ankle impingement syndromes. American Journal of Roentgenology. 2003 Aug;181(2):551-9.</ref><ref>Frigg A, Magerkurth O, Valderrabano V, Ledermann HP, Hintermann B. The effect of osseous ankle configuration on chronic ankle instability. British journal of sports medicine. 2007 Jul 1;41(7):420-4.</ref>


<br>Keywords:<br>• Traumatology<br>• Fractures<br>• Orthopaedics
== Epidemiology/Etiology: ==


<br>Search engines: VUBIS catalogus / UA catalogus<br><br>
This syndrome is called '''footballer's ankle or athlete’s ankle''',because these sports are responsible for creating pressure on the cartilage in the ankle<ref>Cloke DJ, Spencer S, Hodson A, Deehan D. The epidemiology of ankle injuries occurring in English Football Association academies. British journal of sports medicine. 2009 Dec 1;43(14):1119-25.</ref>. This is especially common in football where the kicking motion is responsible for reoccurring micro trauma’s, leading to an increased chance of anterior impingement syndrome (AIS). These 2 sports together with dance are the majority of AIS patient’s.<br>Epidemiological figures were not found but because anterior impingement is frequently preceded by an ankle sprain,'''AIS''' of the ankle is a chronic disorder. Pain occurs in full dorsal flexion or full plantar flexion. AIS does not occur as a result of insidious onset. AIS will usually occur when there is pre-existing damage to the ankle. The cause of this damage can be an inflammation. Due to inflammation the soft tissue will swell and have reduced elasticity, leading to an impingement. The second cause can be arthritis, the body responds to arthritis by building extra bone tissue, these are called osteophytes, the body acts this way because it prevents reoccurring micro trauma’s due to decreased range of movement<ref>Tol JL, van Dijk CN. Etiology of the anterior ankle impingement syndrome: a descriptive anatomical study. Foot & ankle international. 2004 Jun;25(6):382-6.</ref>.<br>  
== Risk Factors:  ==


== Definition/Description  ==
There is an increased chance of developing AIS if there have been one or more sprains in the past. When the ankle has been sprained, the ligaments stretch out and lose the ability to efficiently communicate with the brain. The result of this is decreased coordination of the ankle. Which in turn results in more micro trauma’s and ultimately to a higher possibility of AIS. Sports which require a high frequency of full dorsiflexion often result in a greater risk of developing AIS. These sports are primarily football and running.<ref name=":1">Umans H. Ankle impingement syndromes. InSeminars in musculoskeletal radiology 2002 (Vol. 6, No. 02, pp. 133-140). Copyright© 2002 by Thieme Medical Publishers, Inc., 333 Seventh Avenue, New York, NY 10001, USA. Tel.:+ 1 (212) 584-4662.</ref><br>


add text here <br>
== Diagnostic Procedures:  ==


== Clinically Relevant Anatomy  ==
Diagnosis can be made based on 3 sources of information:<br>


The coccyx, also known as the tailbone, is a small triangular bone that is usually formed by fusion of the four rudimentary coccygeal vertebrae, although in some people there may be one less or one more. The coccygeal vertebra 1 (Co1) may remain separate from the fused group, but with increasing age Co1 often fuses with the sacrum and the remaining coccygeal vertebrae usually fuse to form a single bone. (level of evidence D) <br>The forward movement of coccyx is performed actively by the M. levator ani, and the backward movement of coccyx is passively caused by relaxation of these muscles. The coccyx also provides the site of attachment for the M. gluteus maximus as well as the M. levator ani, which is responsible for voluntary control of bladder and bowel. (level of evidence B) The coccyx does not participate with the other vertebrae in support of body weight when standing, however when sitting it may flex anteriorly, indicating that it’s receiving some weight. ( level of evidence D)i
*Clinical Investigation:


The coccyx is part of the sacrococcygeal joint, an cartilaginous joint with an IV disc. Fibrocartilage and ligaments join the apex of the sacrum to the base of the coccyx. The anterior and posterior sacrococcygeal ligaments are long strands that reinforce the joint. (level of evidence D) <br>The coccyx is also attached with the margin of the anus by the anococcygeal ligament. (level of evidence D)iii<br><br>
X-ray can bring clarity if osteophytes are present. The x-ray should be taken from the lateral side, where the bone structures of the ankle are clearly visible. It is also to bear in mind that some osteophytes still may not be visible on the x-ray. To solve this problem research has shown that an anteromedial impingement recording, this is an x-ray where the angle is changed for optimal osteophyte recognition, is a useful adjunct to the standard x-ray.  


== Epidemiology /Etiology  ==
*Patient Subjective History


Fracture of the coccyx often arise after a fall on the buttock, most prevalent a fall of the stairs on the tailbone, or by an impact directly applied. (level of evidence D) An especially difficult childbirth occasionally injures the mother’s coccyx. ( level of evidence D) <br><br>
Taking a thorough and complete subjective history from the patient can be vitally important in diagnosing a case of AIS. In the subjective assessment, the physiotherapist will gain information on any previous injuries at the ankle and will gain insight into the patient's hobbies and occupation, both influencing factors in the development of AIS.<ref name=":1" />  


== Characteristics/Clinical Presentation  ==
*Patient Objective Assessment


1. GENERAL SYMPTOMS<br>General symptoms that appear are (level of evidence D)iv,vii,viii&nbsp;:<br>• Pain that increases in severity when sitting or getting up from a chair<br>• Provoked pain over the tailbone<br>• Bruising or swelling in the tailbone area<br>• Bowel movements and straining are often painful<br>• There are no neurological signs
During the physiotherapist's objective assessment, they will look for the following:  


#Pain while palpating the anterior side of the joint.
#Pain with forced dorsiflexion.
#Whether it is possible to reproduce anterior impingement pain by palpating the anterolateral ankle in plantar flexion, then dorsiflexing the ankle while maintaining pressure with the examiner’s digit over the anterolateral ankle. An increase in pain is 95% sensitive and 88% specific for AIS.
#AIS test should be positive: standing on both feet and leaning forwards, positive is defined as pain and possibly a difference of approximately 5 degrees between the two ankles.
#Ankle may be swollen and can be red.<br>


== Medical Management:  ==


An important number of people suffer from long lasting pains over the coccyx following trauma (with or without fracture of the coccyx), better known as cocydynia.(level of evidence D)
<br>'''1. Limitation:'''<br>


<br>2. CLINICAL PRESENTATION IN PATIENTS WITH SPINAL CORD INJURY<br>Patients with SCI, suffering from painful symptoms in the low back, gluteal, hip and thigh region, have coccyx fracture with a frequency of 34,6%. Patients who had coccyx fracture have higher pain scores when compared with those who don’t have any fracture, however the difference is only statistical significant regarding Sensory Pain Index (SPI) and total McGill scores.( level of evidence C)
The first advise that has to be given to the patient is that he or she should stop from any activity that increases pain, this allows the body to start the healing process without further tissue damage while at the same time preventing a worsening of the situation and the problem becoming chronic. Later on, activity can gradually start building up before returning to original levels.  


<br>
*Alternative exercises can be used with which generates less force on the ankle, this includes; swimming, cycling, water sport etc.
*Another option is to use a slightly raised heel, this means that the foot does not need to go as far up as normal. The use of crutches is another method to prevent further damage to the ankle<ref>Hawkins RD, Fuller CW. A preliminary assessment of professional footballers' awareness of injury prevention strategies. British journal of sports medicine. 1998 Jun 1;32(2):140-3.</ref>.


== Differential Diagnosis  ==
<br>'''2. Reducing Pain:'''<br>


<br>
*Cryotherapy is an effective way of managing the pain created by AIS. Based on the available evidence, cryotherapy seems to be effective in decreasing pain. In comparison with other rehabilitation techniques, the efficacy of cryotherapy has been questioned.&nbsp; Additionally, the low methodological quality of the available evidence is of concern. Many more high-quality studies are required to create evidence-based guidelines on the use of cryotherapy. These must focus on developing modes, durations, and frequencies of ice application that will optimize outcomes after injury. (Does Cryotherapy Improve Outcomes With Soft Tissue Injury? computer-based literature search Hubbard TJ, Denegar CR. Pennsylvania State University, University Park, PA. evidence level&nbsp;: 1 )
*Anti-inflammatory medication reduces pain and supports the healing process.
*Corticoid injection: An occasional injection of corticosteroids into the ankle joint may be helpful particularly if there is some underlying ankle arthritis present.


<br>
<br>'''3. Surgery:'''<br>  


== Diagnostic Procedures  ==
In some instances of AIS operative treatment may be helpful. If the main cause of a patient’s symptoms is from impingement (rather than ankle arthritis) removing the prominent impinging bony spurs can relieve symptoms. Surgical treatment of ankle impingement involves removing the prominent bone spurs either by arthroscopic surgery or by opening up the ankle joint with an incision.<ref>Liu SH, Raskin A, Osti L, Baber C, Jacobson K, Finerman G. Arthroscopic treatment of anterolateral ankle impingement. Arthroscopy: The Journal of Arthroscopic & Related Surgery. 1994 Apr 1;10(2):215-8.</ref>


1. MEDICAL DIAGNOSIS<br>A plain radiography or MRI is necessary to confirm the diagnose of a coccyx fracture. (level of evidence D)
If the bone spurs are large it is often easier and faster to simply open up the ankle joint and remove the bone spurs rather than attempt to do this arthroscopic. Surgery to remove impinging bone spurs from the front of the ankle will not typically help symptoms if the pain is generalized around the ankle due to significant ankle arthritis rather than specifically located in the front of the ankle.  


<br>2. CLINICAL DIAGNOSIS<br>The diagnose is made after rectal examination. (level of evidence D)iv By passing the finger up the rectum and then pressing the bone backwards and forward, the unnatural degree of motion will then be felt. Related to the age and sex of the patient must be remembered that in the female this bone naturally possesses more motion than in the male, and that in youth a degree of motion, that does not exist at a later period of life, is present, allowing the ossification being less complete. However the free motion of the bone is taken as a symptom. (level of evidence D)
In some instances surgery to remove the bony spurs can make a patient’s symptoms worse if it allows the ankle joint to move more and the ankle joint itself has significant arthritis.The bony spurs themselves will tend to grow back over time. Recurrence of symptoms is not uncommon.At long-term follow-up, arthroscopic excision of both soft-tissue overgrowths and osteophytes was shown to be an effective way of treating anterior impingement, provided that there was no preoperative narrowing of the joint space.<ref>Starzyk M, Kentel M, Czamara A, Królikowska A. O-44 Return to the sport of soccer players after arthroscopic treatment consequences of ankle injury.</ref><br>


<br>
*'''Other Management Options:'''


== Outcome Measures ==
Soft tissue massage can help reduce pain.


add links to outcome measures here (also see [[Outcome Measures|Outcome Measures Database]])
#Use of heat improves the blood circulation and stimulates the healing process<br>
#Taping/Bracing of the ankle helps to control movement and support the natural structure of the ankle<ref>McLean DA. Use of adhesive strapping in sport. British journal of sports medicine. 1989 Sep;23(3):147.</ref>.&nbsp;<ref>Verbrugge JD. The effects of semirigid Air-Stirrup bracing vs. adhesive ankle taping on motor performance. Journal of orthopaedic & sports physical therapy. 1996 May;23(5):320-5.</ref><br>


== Examination  ==
These methods are questionable as there is a lack of detailed research supporting their use meaning it is not supported by evidence based practice, one of the core principles of physiotherapy.&nbsp;<br>


add text here related to physical examination and assessment<br>  
== Physical Therapy Management:<ref>Balduini FC, Vegso JJ, Torg JS, Torg E. Management and rehabilitation of ligamentous injuries to the ankle. Sports Medicine. 1987 Sep 1;4(5):364-80.</ref>   ==


== Medical Management <br>  ==
Physiotherapy can be given following the KNGF guidelines, which are complementary with the different phases of inflammation: &nbsp; &nbsp;


1. COCCYGEOPLASTY<br>By applying the novel techniques that are used in vertebroplasty and sacroplasty, coccygeoplasty is introduced as a new percutaneous treatment modality for fractures of the coccyx. This procedure can be helpful for patients with refractory pain resulting from a fracture of the coccyx and can be performed quickly and safely with high-resolution c-arm fluoroscopy. The coccygeal fracture treated with an injection of polymethylmethacrylate cement can provide early symptom relief. Although the promising results, an experience with a larger patient population is warranted. ( level of evidence C)
<br>'''Phase 1 of recovery''': inflammation 0-3 days<br>


<br>2. COCCYGECTOMY<br>Literature reports suggest that coccygectomy, partial or total removal of the coccyx, has been beneficial with success rates as high as 60-91%. However, coccygectomy is a more invasive procedure, with a common complication rate as high as 22%, and is usually associated with perineal contamination of the wound. Other complications could include persistent bleeding from the hemorrhoidal venous complex of the rectum. (level of evidence C)ix
*Rest and immobilization, but frequent movement within pain limits.
*Use pain as a guideline to base you’re exercises on.  
*Eating behaviours (vitamin C, proteins, anti-oxidants,…).  
*Exercise basic functions, move feet and toes to improve blood circulation.  
*Limit painkillers to the bare minimum.


<br>
'''<br>Phase 2 of recovery''': limited functionality &nbsp;4-10 days<br>  


== Physical Therapy Management <br>  ==
*Mobilization to improve scar tissue quality.
*Muscle endurance training within free ROM.
*Proprioceptive training.
*Train muscle strength of “foot raiser muscles” (tricep surae muscles or the plantar flexor muscles. The gastrocnemius and soleus muscles) using more weight and less repetitions.
*Normal ROM should be reached.


add text here <br>  
'''<br>Phase 3 of recovery''': early revalidation 11-21 days<br>  


== Key Research ==
*Mobilizations with a progressively enhancing ROM
*Proprioceptive training
*Musclestrenght training
*Research and estimate the level of daily activity, modify therapy with this info


add links and reviews of high quality evidence here (case studies should be added on new pages using the [[Template:Case Study|case study template]])<br>  
<br>'''Phase 4 of recovery''': late revalidation 3-6 weeks<br>  


== Resources <br>  ==
*Improve load capacity, running and climbing stairs.
*Coordination training with ADL.
*Goal is to end exercises with a load like before the incident.
*Progressivebuild up of schedule&nbsp;: static  dynamic / selective  functional.


add appropriate resources here <br>  
<br>'''Phase 5 of recovery:''' early sporting: 6-8 weeks<br>  


== Clinical Bottom Line  ==
'''Phase 6 of recovery''': late sporting starting week 8<br>


add text here <br>
*Improve sporting.
*Practice and improve functions and activities based on sports.
*Sufficient rest and repair moments.
'''Prevention:'''


== Recent Related Research (from [http://www.ncbi.nlm.nih.gov/pubmed/ Pubmed]) ==
It is recommended to improve the  awareness of injury prevention strategies for '''players and athletes.''' These strategies  were identified as:  
* use of shin pads during training,
* carbohydrate intake before and after training and after matches,
* cool downs after training and matches, and flexibility work.<ref>Hawkins RD, Fuller CW. A preliminary assessment of professional footballers' awareness of injury prevention strategies. British journal of sports medicine. 1998 Jun 1;32(2):140-3.</ref>  


see tutorial on [[Adding PubMed Feed|Adding PubMed Feed]]
== References:  ==
<div class="researchbox">
<references /> <br>
<rss>Feed goes here!!|charset=UTF-8|short|max=10</rss>  
</div>  
== References  ==


see [[Adding References|adding references tutorial]].
[[Category:Conditions]]  
 
[[Category:Ankle]]
<references />
[[Category:Joints]]  
 
[[Category:Ankle - Conditions]]
[[Category:Vrije_Universiteit_Brussel_Project|Template:VUB]]
[[Category:Musculoskeletal/Orthopaedics]]
 
[[Category:Sports Medicine]]
MOORE K.L., DALLEY A.F., AGUR A.M.R., Clinically oriented anatomy: chapter 3: Pelvis and perineum, Wolters Kluwer health, sixth edition, 2010, pag. 451-452, level of evidence D<br> YU-TSAI T., LI-WEN T., CHENG-HSIU L., SHIH-WEI C., The influence of human coccyx in body weight shifting, medicine and science in sport and exercise, 2011, Volume 43, Number 5, pag. 494-496, level of evidence B<br> MOORE K.L., DALLEY A.F., AGUR A.M.R., Clinically oriented anatomy: chapter 3: Pelvis and perineum, Wolters Kluwer health, sixth edition, 2010, pag. 332, level of evidence D<br> HAARMAN H.J.Th.M., Klinische traumatologie, Elsevier gezondheidszorg, 2006, pag. 117, level of evidence D<br> MOORE K.L., DALLEY A.F., AGUR A.M.R., Clinically oriented anatomy: chapter 3: Pelvis and perineum, Wolters Kluwer health, sixth edition, 2010, pag. 461, level of evidence D<br> TEKIN L. et al., Coccyx fracture in patients with spinal cord injury, European journal of physical and rehabilitation medicine, March 2010, Volume 46, Number 1, pag. 43-46, level of evidence C<br> RAISSAKI M.T.,Fracture dislocation of the sacro-coccygeal joint: MRI evaluation, Pediatric radiology, March 1999, pag. 642-643, level of evidence D<br> LONSDALE E.F., A practical treatise on fractures, Walton and Mitchell printers, 1838, pag. 269-270, level of evidence D<br> MIYAMOTO K. et al., Exposure to pulsed low intensity ultrasound stimulates extracellular matrix metabolism of bovine intervertebral dosc cells cultured in alginate beads, Spine, November 2005, level of evidence B<br> EBNEZAR J., Essentials of orthopaedics for physiotherapist, Jaypee, 2003, pag. 174, level of evidence D<br> DEAN L.M. et al., Coccygeoplasty : treatment for fractures of the coccyx, J. Vasc. Interv. Radiol, 2006, pag. 909-912, level of evidence C<br> COOPER G., HERRERA J.E., Manual of musculoskeletal medicine, Wolters kluwer, Lippincott Williams &amp; Wilkins, 2008, pag. 144, level of evidence D&amp;nbsp;
[[Category:Sports Injuries]]

Latest revision as of 12:33, 23 August 2019

Definition/Description:[edit | edit source]

The anterior impingement syndrome of the ankle is a strangulation that can be caused by soft tissue, like the joint capsule or scar tissue, and hard tissue which refers to bone tissue. It’s location is the anterior side of the ankle in the talocrural joint. Due to repeated micro trauma’s little the ankle, the body will respond to this by building extra bone tissue called “osteophytes”. There are 3 types of osteophytes:

  • Traction spur.
  • Inflammatory spur.
  • Osteophyte or chondro-osteophyte.

The one occurring here is a chondro-osteophyte.It is a defense mechanism of the body that prevents further injury, but reduces mobility and can lead to pain due to an impingement. There may potentially be swelling of the capsule and connective tissue.[1] The relative contributions of the osseous and soft-tissue abnormalities are variable, but whatever component is dominant there physical impingement and painful limitation of ankle movement.[2]

Clinically Relevant Anatomy:[edit | edit source]

  • The joint capsule is ventral and dorsal minimally present, it is reinforced with ligaments the lateral side. At the the tendons of the lower leg muscles, at the back is the tendon of the medial flexor . These muscles prevent the parts of the joint capsule from getting trapped between bones.
    Ank1.jpg
  • Foot free: In dorsiflexion the distal end of the talus moves lateral and the plantar side of the talus rotates to lateral. In plantar flexion the distal end of the talus moves medial, the plantar side of the talus rotates to medial.
  •  Foot stabilized on the floor: dorsal flexion provokes of the tibia with the distal end of the tibia moves to medial. Plantar flexion provokes an exorotation of the tibia with the distal end of the tibia moves to lateral Stabilityin the talocrural joint is the weakest in neutral position, this is because in this position the ligaments that surround the ankle are less tensed than in dorsal flexion or plantar flexion and there is less contact between joint facets. Plantar flexion has more stability, there is more contact between the joint facets and the tibionavicular part as well as the tibiotalar anterior part of the ligament deltoideum. The greatest stability occurs in dorsiflexion, the large front part of the trochlea tali now comes in touch with the small, narrow back part of joint socket, when this happens, the tibia and the fibula widen a bit and keep the talus closely bound helped with the strong tibiofibular ligaments.[1][3]

Epidemiology/Etiology:[edit | edit source]

This syndrome is called footballer's ankle or athlete’s ankle,because these sports are responsible for creating pressure on the cartilage in the ankle[4]. This is especially common in football where the kicking motion is responsible for reoccurring micro trauma’s, leading to an increased chance of anterior impingement syndrome (AIS). These 2 sports together with dance are the majority of AIS patient’s.
Epidemiological figures were not found but because anterior impingement is frequently preceded by an ankle sprain,AIS of the ankle is a chronic disorder. Pain occurs in full dorsal flexion or full plantar flexion. AIS does not occur as a result of insidious onset. AIS will usually occur when there is pre-existing damage to the ankle. The cause of this damage can be an inflammation. Due to inflammation the soft tissue will swell and have reduced elasticity, leading to an impingement. The second cause can be arthritis, the body responds to arthritis by building extra bone tissue, these are called osteophytes, the body acts this way because it prevents reoccurring micro trauma’s due to decreased range of movement[5].

Risk Factors:[edit | edit source]

There is an increased chance of developing AIS if there have been one or more sprains in the past. When the ankle has been sprained, the ligaments stretch out and lose the ability to efficiently communicate with the brain. The result of this is decreased coordination of the ankle. Which in turn results in more micro trauma’s and ultimately to a higher possibility of AIS. Sports which require a high frequency of full dorsiflexion often result in a greater risk of developing AIS. These sports are primarily football and running.[6]

Diagnostic Procedures:[edit | edit source]

Diagnosis can be made based on 3 sources of information:

  • Clinical Investigation:

X-ray can bring clarity if osteophytes are present. The x-ray should be taken from the lateral side, where the bone structures of the ankle are clearly visible. It is also to bear in mind that some osteophytes still may not be visible on the x-ray. To solve this problem research has shown that an anteromedial impingement recording, this is an x-ray where the angle is changed for optimal osteophyte recognition, is a useful adjunct to the standard x-ray.

  • Patient Subjective History

Taking a thorough and complete subjective history from the patient can be vitally important in diagnosing a case of AIS. In the subjective assessment, the physiotherapist will gain information on any previous injuries at the ankle and will gain insight into the patient's hobbies and occupation, both influencing factors in the development of AIS.[6]

  • Patient Objective Assessment

During the physiotherapist's objective assessment, they will look for the following:

  1. Pain while palpating the anterior side of the joint.
  2. Pain with forced dorsiflexion.
  3. Whether it is possible to reproduce anterior impingement pain by palpating the anterolateral ankle in plantar flexion, then dorsiflexing the ankle while maintaining pressure with the examiner’s digit over the anterolateral ankle. An increase in pain is 95% sensitive and 88% specific for AIS.
  4. AIS test should be positive: standing on both feet and leaning forwards, positive is defined as pain and possibly a difference of approximately 5 degrees between the two ankles.
  5. Ankle may be swollen and can be red.

Medical Management:[edit | edit source]


1. Limitation:

The first advise that has to be given to the patient is that he or she should stop from any activity that increases pain, this allows the body to start the healing process without further tissue damage while at the same time preventing a worsening of the situation and the problem becoming chronic. Later on, activity can gradually start building up before returning to original levels.

  • Alternative exercises can be used with which generates less force on the ankle, this includes; swimming, cycling, water sport etc.
  • Another option is to use a slightly raised heel, this means that the foot does not need to go as far up as normal. The use of crutches is another method to prevent further damage to the ankle[7].


2. Reducing Pain:

  • Cryotherapy is an effective way of managing the pain created by AIS. Based on the available evidence, cryotherapy seems to be effective in decreasing pain. In comparison with other rehabilitation techniques, the efficacy of cryotherapy has been questioned.  Additionally, the low methodological quality of the available evidence is of concern. Many more high-quality studies are required to create evidence-based guidelines on the use of cryotherapy. These must focus on developing modes, durations, and frequencies of ice application that will optimize outcomes after injury. (Does Cryotherapy Improve Outcomes With Soft Tissue Injury? computer-based literature search Hubbard TJ, Denegar CR. Pennsylvania State University, University Park, PA. evidence level : 1 )
  • Anti-inflammatory medication reduces pain and supports the healing process.
  • Corticoid injection: An occasional injection of corticosteroids into the ankle joint may be helpful particularly if there is some underlying ankle arthritis present.


3. Surgery:

In some instances of AIS operative treatment may be helpful. If the main cause of a patient’s symptoms is from impingement (rather than ankle arthritis) removing the prominent impinging bony spurs can relieve symptoms. Surgical treatment of ankle impingement involves removing the prominent bone spurs either by arthroscopic surgery or by opening up the ankle joint with an incision.[8]

If the bone spurs are large it is often easier and faster to simply open up the ankle joint and remove the bone spurs rather than attempt to do this arthroscopic. Surgery to remove impinging bone spurs from the front of the ankle will not typically help symptoms if the pain is generalized around the ankle due to significant ankle arthritis rather than specifically located in the front of the ankle.

In some instances surgery to remove the bony spurs can make a patient’s symptoms worse if it allows the ankle joint to move more and the ankle joint itself has significant arthritis.The bony spurs themselves will tend to grow back over time. Recurrence of symptoms is not uncommon.At long-term follow-up, arthroscopic excision of both soft-tissue overgrowths and osteophytes was shown to be an effective way of treating anterior impingement, provided that there was no preoperative narrowing of the joint space.[9]

  • Other Management Options:

Soft tissue massage can help reduce pain.

  1. Use of heat improves the blood circulation and stimulates the healing process
  2. Taping/Bracing of the ankle helps to control movement and support the natural structure of the ankle[10][11]

These methods are questionable as there is a lack of detailed research supporting their use meaning it is not supported by evidence based practice, one of the core principles of physiotherapy. 

Physical Therapy Management:[12][edit | edit source]

Physiotherapy can be given following the KNGF guidelines, which are complementary with the different phases of inflammation:    


Phase 1 of recovery: inflammation 0-3 days

  • Rest and immobilization, but frequent movement within pain limits.
  • Use pain as a guideline to base you’re exercises on.
  • Eating behaviours (vitamin C, proteins, anti-oxidants,…).
  • Exercise basic functions, move feet and toes to improve blood circulation.
  • Limit painkillers to the bare minimum.


Phase 2 of recovery
: limited functionality  4-10 days

  • Mobilization to improve scar tissue quality.
  • Muscle endurance training within free ROM.
  • Proprioceptive training.
  • Train muscle strength of “foot raiser muscles” (tricep surae muscles or the plantar flexor muscles. The gastrocnemius and soleus muscles) using more weight and less repetitions.
  • Normal ROM should be reached.


Phase 3 of recovery
: early revalidation 11-21 days

  • Mobilizations with a progressively enhancing ROM
  • Proprioceptive training
  • Musclestrenght training
  • Research and estimate the level of daily activity, modify therapy with this info


Phase 4 of recovery: late revalidation 3-6 weeks

  • Improve load capacity, running and climbing stairs.
  • Coordination training with ADL.
  • Goal is to end exercises with a load like before the incident.
  • Progressivebuild up of schedule : static  dynamic / selective  functional.


Phase 5 of recovery: early sporting: 6-8 weeks

Phase 6 of recovery: late sporting starting week 8

  • Improve sporting.
  • Practice and improve functions and activities based on sports.
  • Sufficient rest and repair moments.

Prevention:

It is recommended to improve the awareness of injury prevention strategies for players and athletes. These strategies were identified as:

  • use of shin pads during training,
  • carbohydrate intake before and after training and after matches,
  • cool downs after training and matches, and flexibility work.[13]

References:[edit | edit source]

  1. 1.0 1.1 Cerezal L, Abascal F, Canga A, Pereda T, García-Valtuille R, Pérez-Carro L, Cruz A. MR imaging of ankle impingement syndromes. American Journal of Roentgenology. 2003 Aug;181(2):551-9.
  2. Cerezal L, Abascal F, Canga A, Pereda T, García-Valtuille R, Pérez-Carro L, Cruz A. MR imaging of ankle impingement syndromes. American Journal of Roentgenology. 2003 Aug;181(2):551-9.
  3. Frigg A, Magerkurth O, Valderrabano V, Ledermann HP, Hintermann B. The effect of osseous ankle configuration on chronic ankle instability. British journal of sports medicine. 2007 Jul 1;41(7):420-4.
  4. Cloke DJ, Spencer S, Hodson A, Deehan D. The epidemiology of ankle injuries occurring in English Football Association academies. British journal of sports medicine. 2009 Dec 1;43(14):1119-25.
  5. Tol JL, van Dijk CN. Etiology of the anterior ankle impingement syndrome: a descriptive anatomical study. Foot & ankle international. 2004 Jun;25(6):382-6.
  6. 6.0 6.1 Umans H. Ankle impingement syndromes. InSeminars in musculoskeletal radiology 2002 (Vol. 6, No. 02, pp. 133-140). Copyright© 2002 by Thieme Medical Publishers, Inc., 333 Seventh Avenue, New York, NY 10001, USA. Tel.:+ 1 (212) 584-4662.
  7. Hawkins RD, Fuller CW. A preliminary assessment of professional footballers' awareness of injury prevention strategies. British journal of sports medicine. 1998 Jun 1;32(2):140-3.
  8. Liu SH, Raskin A, Osti L, Baber C, Jacobson K, Finerman G. Arthroscopic treatment of anterolateral ankle impingement. Arthroscopy: The Journal of Arthroscopic & Related Surgery. 1994 Apr 1;10(2):215-8.
  9. Starzyk M, Kentel M, Czamara A, Królikowska A. O-44 Return to the sport of soccer players after arthroscopic treatment consequences of ankle injury.
  10. McLean DA. Use of adhesive strapping in sport. British journal of sports medicine. 1989 Sep;23(3):147.
  11. Verbrugge JD. The effects of semirigid Air-Stirrup bracing vs. adhesive ankle taping on motor performance. Journal of orthopaedic & sports physical therapy. 1996 May;23(5):320-5.
  12. Balduini FC, Vegso JJ, Torg JS, Torg E. Management and rehabilitation of ligamentous injuries to the ankle. Sports Medicine. 1987 Sep 1;4(5):364-80.
  13. Hawkins RD, Fuller CW. A preliminary assessment of professional footballers' awareness of injury prevention strategies. British journal of sports medicine. 1998 Jun 1;32(2):140-3.