Graves' Disease: Difference between revisions
Erin Shinkle (talk | contribs) No edit summary |
Erin Shinkle (talk | contribs) No edit summary |
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== Prevalence == | == Prevalence == | ||
Graves’ disease is more prevalent in the Caucasian race | Graves’ disease is more prevalent in the Caucasian race affecting more women than men at a ratio of 4:1 and ages range from 30-60years (Hemmiki and DD). Graves’ disease accounts for 85% of all cases of hyperthyroidism. (Pathology) | ||
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Revision as of 23:07, 29 March 2011
Original Editors - Erin Shinkle from Bellarmine University's Pathophysiology of Complex Patient Problems project.
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Definition/Description[edit | edit source]
Graves’ is classified as an autoimmune disease that affects the thyroid gland. It causes goiters, hyperthyroidism, ophthalmopathy, and occasionally dermopathy.(ginsberg) Graves' disease is known for increasing the thyroid stimulating hormone raising the T4 levels and is known as the number one cause of hyperthyroidism.(DD) Hyperthyroidism/Graves' disease can manifest itself in multiple systems(link to hyperthyroidism page).
Prevalence[edit | edit source]
Graves’ disease is more prevalent in the Caucasian race affecting more women than men at a ratio of 4:1 and ages range from 30-60years (Hemmiki and DD). Graves’ disease accounts for 85% of all cases of hyperthyroidism. (Pathology)
Characteristics/Clinical Presentation[edit | edit source]
Goiter, Exophthalmos, tremors, tachycardia with palpitations, heat intolerances, weight loss, increased deep tendon reflexes, weakness and muscle atrophy, increased cardiac output, myasthenia gravis, thin hair, warm moist skin, sensitivity to light, dysphasia, diarrhea, amenorrhea, polyuria, and many other presentations some of which may not be as common and more subtle. (pathology)
Associated Co-morbidities[edit | edit source]
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Medications[edit | edit source]
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Diagnostic Tests/Lab Tests/Lab Values[edit | edit source]
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Etiology/Causes[edit | edit source]
Has both a genetic, lack of suppressor t-cells causing increase in TSH receptor antibodies, and environmental causes which includes, but not limited to the following: stress, smoking, post pardum, and infections (Hemminki and ginsburg)
Systemic Involvement[edit | edit source]
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Medical Management (current best evidence)
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The current best management of Graves' disease varies upon several factors of the individual recieving it. The options include partial and full removal of thyroid gland, antithyroid drug therapy, radioiodine therapy (franklyn).
Physical Therapy Management (current best evidence)[edit | edit source]
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Alternative/Holistic Management (current best evidence)[edit | edit source]
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Differential Diagnosis[edit | edit source]
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Case Reports/ Case Studies[edit | edit source]
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Resources
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National Graves' Disease Foundation
American Thyroid Association (ATA)
http://thyroid.org/
American Association of Clinical Endocrinologists (AACE)
http://www.aace.com/
The Endocrine Society
http://www.endo-society.org/
The Hormone Foundation
http://www.hormone.org/
American Autoimmune Related Diseases Associations (AARDA)
Recent Related Research (from Pubmed)[edit | edit source]
Recent Related Research (from Pubmed)[edit | edit source]
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References[edit | edit source]
see adding references tutorial.