Snapping Hip and Trochanteric Bursitis: Difference between revisions

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   <div class="col-md-6"> {{#ev:youtube|v=XCOD1dkcVk0|250}} <div class="text-right"><ref>FFITT Health Global. FABER Test. 2010.Available from: https://www.youtube.com/watch?v=XCOD1dkcVk0 [last accessed 15/03/2022]</ref></div></div>
   <div class="col-md-6"> {{#ev:youtube|v=XCOD1dkcVk0&t=43s|250}} <div class="text-right"><ref>FFITT Health Global. FABER Test. 2010.Available from: https://www.youtube.com/watch?v=XCOD1dkcVk0&t=43s [last accessed 15/03/2022]</ref></div></div>
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AIF Education. Functional Movement Test - Step Up and Down. 2013.Available from: https://www.youtube.com/watch?v=uUUDSxcO5VY[last accessed 15/03/2022]</ref></div></div>
AIF Education. Functional Movement Test - Step Up and Down. 2013.Available from: https://www.youtube.com/watch?v=uUUDSxcO5VY[last accessed 15/03/2022]</ref></div></div>

Revision as of 15:07, 15 March 2022

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Snapping Hip Syndrome (SHS)[edit | edit source]

There are three types of the Snapping Hip syndromes identified: the external, the internal, and the intra-articular snapping hip.

External Snapping Hip Syndrome (ESHS)[edit | edit source]

External (lateral) snapping hip is a painful condition also known as lateral coxa saltans. It occurs in about 10% of the general population.[1] The external extra-articular factor causing the ESHS involves the posterior iliotibial band snapping over the greater trochanter. [2] Second external extra-articular mechanism leading to this condition involves the anterior aspect of the gluteus maximus travelling over the greater trochanter during hip flexion and extension or internal and external rotation of the hip and snapping over the greater trochanter. This may lead to the inflammation of the trochanteric bursa. [2]Individuals with the ESHS may also have coxa vara, fibrotic scar tissue, a prominent greater trochanter, smaller lateral pelvic width, or a past surgery for anterolateral knee instability.[3][4]

Internal Snapping Hip Syndrome (ISHS)[edit | edit source]

Internal extra-articular snapping is caused by the iliopsoas tendon flipping over the iliopectinal eminence or the femoral head and often requires hip flexors contraction. Other proposed extra-articular mechanisms of this condition include:

  • accessory iliopsoas tendon slips
  • iliopsoas snapping over a ridge at the lesser trochanter
  • snapping of the iliofemoral ligament over the femoral head
  • subluxation of the long head of the biceps at the ischium
  • snapping at the anterior inferior iliac spine.[2]

Intra-Articular Snapping Hip[edit | edit source]

The intra-articular factors can lead to the development of snapping hip symptoms. They include labral tears, ligamentum teres tears, loose bodies such as osteochondral fragments, or joint instability.

Clinically Relevant Anatomy[edit | edit source]

Iliotibial Band[edit | edit source]

The iliotibial band (ITB) is a thick band of fascia that runs on the lateral side of the thigh from the iliac crest and inserts at the knee.[5] It is composed of dense fibrous connective tissue that originates from the anterior fibres of the tensor fasciae late and the posterior fibres of the gluteus maximus. After running down on the lateral thigh it ends with most of the fibres inserting onto the tibial tubercle, some inserting into the gluteus maximum tendon, the lateral femoral epicondyle and distal lateral border of the patella. [1]During hip flexion, the ITB moves anterior to the greater trochanter and during hip extension it runs posterior to it. [2]

On the lateral aspect of the hip joint, there are three subgluteal bursae: greater trochanter bursa, subgluteus medius bursa, and subgluteus minimus bursa. The trochanteric bursa is the largest of the three and it covers the insertion of the gluteus medius tendon and the posterior aspect of the greater trochanter. Its primary role is to reduce friction between the tendons of the gluteus medius and the ITB when they pass over the greater trochanter.[6]

The iliotibial band role includes hip stabilisation during a single leg stance and ambulation when the band acts as a tendon of the tensor fasciae latae (TFL) and the gluteus maximus. The contraction of the gluteus maximus and the TFL muscles cause the tightening of the band.

Gluteus Maximus[edit | edit source]

Gluteus maximus originates at the outer slope of the dorsal segment of the iliac crest, the gluteal surface of the ilium, dorsal surface of the lower part of the sacrum, side of the coccyx, and the sacrotuberous ligament. It also shares its attachment to the thoracolumbar fascia with the ipsilateral multifidus and contralateral latissimus dorsi. The muscle inserts onto gluteal tuberosity and the iliotibial band.

Gluteus Maximus's primary role includes hip extension, external rotation and abduction. Its function is essential to maintain an erect posture.

Iliopsoas[edit | edit source]

Iliopsoas muscle complex is made up of three muscles that include the iliacus, psoas major and psoas minor.[2] It can work as a unit, but each of the three muscles has separate functions. The iliopsoas originates at the fusion of the psoas major and iliacus muscles. This fusion occurs at the level of L5-S2, and the combined muscles pass from the pelvis to the thigh under the inguinal ligament. The iliopsoas muscle inserts into the lesser trochanter of the femur via the psoas tendon.

The iliopsoas bursa is the largest bursa of the hip joint and is located deep to the iliopsoas musculotendinous junction and anterior to the hip joint capsule. The bursa has been reported to communicate with the hip joint in ~15% of patients. It helps the muscle glide and slides over the front of the hip during movement.[7]

The iliopsoas muscle is the strongest hip flexor and assists in the external rotation of the femur. It is essential for correct standing or sitting lumbar posture, and during walking and running.

Pathophysiology[edit | edit source]

  • External Snapping Hip
    • visual component of the snap observed by the patient when the ITB slides from the anterior to posterior aspect of the greater trochanter during hip movement from extension to flexion. This is usually caused by the thickening of the posterior ITB and/or anterior portion of the gluteus maximus.[8]
  • Internal Snapping Hip
    • audible component of the snap when the iliopsoas tendon movement from the lateral to the medial position is restricted during hip extension and internal rotation. This restriction can be caused by the femoral head or pectineal eminence of the pelvis.[8]
  • Intra-articular Snapping Hip
    • pathological process of the hip joint when loose bodies, acetabular labrum tears, or osteochondral fractures can interfere with the articulation of the femoral head into the acetabulum. [8]

Clinical Presentation[edit | edit source]

The External Snapping Hip syndrome presents with the sensation described as a feeling of hip dislocation. With the Internal Snapping Hip patients describe the feeling of the hip joint being "stuck" or locked and they report hearing the sound of snapping. The intermittent sound of clicking or catching characterises the Intra-articular pathology.

Additionally patients may report:

  • pain
  • muscles weakness
  • activities limitation.[3]

Evaluation[edit | edit source]

Physiotherapy Assessment[edit | edit source]

Physiotherapy assessment includes the patient's history, physical examination and special tests.

Patient's History[edit | edit source]
  • symptoms description (location, sensation, timing and duration)
  • impact on activities of daily living
Physical Examination[edit | edit source]
  • Joint palpation: snapping palpated over the anterior hip indicates coxa saltans interna
  • Range of motion assessment
  • Strength assessment
  • Pain assessment (Visual Analogue Scale)
  • Functional assessment including gait assessment.
Special Tests[edit | edit source]

External Snapping Hip Syndrome:

  • FADIR test including flexion, adduction and internal rotation or impingement
  • Ober test: assessment of the ITB tightness
  • Hip active flexion followed by passive extension and abduction: positive test reproduces the snapping over the greater trochanter.
  • Hula hoop test is an alternative test when the hip is adducted and circumducted in a standing position. Positive test: snapping is reproduced over the greater trochanter
  • Hip active flexion followed by passive extension and abduction: positive test reproduces the snapping over the greater trochanter.
  • Biomechanical abnormalities (Trendelenburg sign)


Internal Snapping Hip Syndrome:

  • Dynamic testing (loop-the-loop): starting at FABER (flexion, abduction and external rotation) position and moving into EADIR (extension, adduction and internal rotation) position. Positive test: snapping of the iliopsoas tendon
  • Thomas test: positive test indicates psoas contracture
  • Stinchfield Test: The patient lies supine with the hip flexed at 30°. The patient is asked to fully flex the hip, while the examines apply a resistance force. Pain in the anterior groin indicates a positive test.


[11]

Imaging[edit | edit source]

  • X-Ray: no typical findings indicative of coxa saltans interna on X-Ray. Evidence of femoroacetabular impingement can be found on a plain X-ray.
  • MRI: not specific for the iliopsoas involvement. May assist with the diagnosis of iliopsoas bursa or iliopsoas muscle inflammation. This modality helps to visualise a thickened ITB and focal thickening of the gluteus maximus when ESH is suspected.[2]
  • Ultrasound: Helps to visualise the dynamic motion of the iliopsoas tendon, but requires a skilled clinician to perform and interpret results. [2][12]
  • Dynamic sonography: able to detect the movement of the ITB over the greater trochanter
  • Iliopsoas bursography: under fluoroscopy bursa is filled with contrast to visualised the tendon moving back and forth. This imaging is performed to confirm ISHS syndrome.[2]

Management / Interventions[edit | edit source]

Conservative Management[edit | edit source]

Goal: to improve pain, to increase flexibility and to equalise the limb length discrepancy if needed.

Conservative approach should be continued for at least 6 months and the following approaches are recommended:[1]

  1. Muscle tightness causing the snapping must be identified: passive and active stretching program specific to the ITB and the iliopsoas muscles to be initiated [2]
  2. Rest, icing and anti-inflammatories should be advocated to reduce inflammation[2]
  3. Movements that provoke the snap should be avoided
  4. Sports activities must be reduced
  5. Active Release Techniques[13]
  6. Use of modalities: Laser therapy or Extracorporeal shockwave therapy ESWT[14]

Surgical Intervention[edit | edit source]

Goal: to release the contracted ITB or the iliopsoas to resolve the snap[15]

External Snapping Hip:

  1. Fractional lengthening of the tendon
  2. Complete release of the tendinous portion[16]

The lengthening procedures include: Z-shaped release, formal Z-lengthening, cross-shaped release and release of the gluteus maximus tendon insertion to the femur.[2][1]

Internal Snapping Hip:

  1. Anterior approach to release the posteromedial tendinous portion of the iliopsoas
  2. Endoscopic release of the iliopsoas tendon[2][17]

Trochanteric Bursitis[edit | edit source]

Lateral hip pain due to the inflammation of the bursa usually as a consequence of microtraumas produces a condition called trochanteric bursitis or greater trochanteric pain syndrome (GTPS).[18][19] It affects females between the age of 40 to 60 most commonly.[20]The abnormal hip biomechanics is the leading cause of development of the symptoms where the gluteus medius and minimus tendinopathy along with or without bursitis can lead to this painful lateral hip syndrome.[20]

Clinically Relevant Anatomy[edit | edit source]

A bursa or bursae (plural form) it is a membrane sac lined by a synovial membrane or synovium and containing the synovial fluid.[21] It is located between bone and surrounding soft tissue and near a joint.[21] Its role is to lubricate and reduce tension and friction between the soft tissue/bone interface. It also acts as a shock absorber during the movement of muscles and joints.  Detailed information on the mechanism of injury or the pathological process of bursitis you can find here.

In the case of Trochanteric Bursitis, two bursae are commonly involved:

  1. Subgluteus Medius bursa - located above the greater trochanter and underneath the insertion of the gluteus medius.
  2. Subgluteus Maximus bursa - located between the greater trochanter and the insertion of the gluteus medius and gluteus maximus muscles.

Pathophysiology[edit | edit source]

Altered lower extremity biomechanics and imbalances lead to the development of the GTPS. The aetiology of this syndrome is multifactorial[19] and the following issues are believed to contribute to its presence:

  • A lower femoral neck-shaft angle [22][23]
  • Recently increased activities levels [22][24]
  • Improper footwear[22]
  • Hormonal changes after menopause [22][25]
  • Degeneration or tendinopathy in the gluteus medius and minimus musculature.[26]
  • Repetitive microtrauma to the bursa caused by friction between the greater trochanter and iliotibial band[27]

Clinical Presentation[edit | edit source]

The following clinical presentation may occur:

  • pain at the lateral side of the hip radiating down[20]
  • difficulties with side-lying on the affected side[20]
  • pain while walking[20]
  • gait abnormalities [20]
  • tenderness over the greater trochanter[24]
  • pain interfering with sleep and physical function[24]

Differential Diagnosis:

Evaluation[edit | edit source]

Physiotherapy Assessment[edit | edit source]

Physiotherapy assessment includes the patient's history, physical examination and special tests.

Patient's History[edit | edit source]
Physical Examination[edit | edit source]
  • Observation
    • Observe patient's posture in sitting and standing: watch for slightly flexed posture, slouch position, leaning to uninvolved side[28]
    • Watch a patient walk: look at pelvic obliquity, the hip tendency to move into adduction while ambulating[20]

[29]

  • Palpation
    • Palpate the iliotibial band and watch for tightness, irritation, pain, trigger points[20]
    • Check the lumbar spine, sacroiliac joints, ischium, iliac crest, lateral aspect of the greater trochanteric bursa, muscle bellies and the pubic symphysis to determine a potential source of hip symptoms or pain[28]
  • Range of motion assessment: check active hip flexion, internal and external rotation, abduction and adduction. Movement can reproduce pain in the injured area.
  • Muscle strength assessment: assess major muscle groups acting on the hip joint. Hip abductor weakness is a common finding and testing the abductors can provoke lateral hip pain during the examination.
  • Functional assessment: mobility, gait. Look for any limb length discrepancy, weakness and heel strike which contributes to the function of the gluteus maximus.[30]
Special Tests[edit | edit source]
  • Jump Sign: patient in side-lying, so the affected side is on top, apply pressure around the lateral hip or the greater trochanteric region. [20]This test a positive predictive value of about 83% for confirmation for the MRI.[20]
  • Single leg stance test: patient to stand for 30 seconds on one leg. The test is positive when the patient is not able to hold the balance. This test has almost a hundred per cent positive predictive value that confirms the MRI findings.[20]
  • FADIR test
  • FABER test
  • Ober's test
  • Step Down and Step Up test
  • Trendelenburg test


Imaging[edit | edit source]

Management/Interventions[edit | edit source]

Conservative Management[edit | edit source]

Goal:

activity modification, ice, oral NSAIDs (non-steroidal anti-inflammatory drugs), and corticosteroid and platelet-rich plasma (PRP) injections.While prior investigations reported that 90% of patients with GTPS experienced pain relief with two years of non-operative management, more recent literature reports that only about 64% of patients find relief after one year and 71% after five years in primary care setting. Additionally, GTPS symptoms often recur and require several rounds of conservative therapy.

So our acute phase management is obviously rest, ice, some soft tissue therapy. Don't go too deep into it. Just superficial release, if we can. Taping, medications such as NSAIDs, paracetamol, painkillers, and your advice for the runner would be first of all to reduce the intensity and frequency of running as much as possible and not to run on uneven surfaces or banked tracks.

Shockwave therapy may be used under special circumstances, and there's not enough evidence as far as ultrasound therapy is concerned. As mentioned before we have exercise and load management obviously, strengthening of the abductors of the hip. And we talked about. Target the adductors, stretch them out, strengthen the abductors on the other side, not forgetting the lumbopelvic control as well. So we are bringing everything back into balance, back into the right biomechanical performance. Now, as much as we want, we know that the hip goes into adduction, which is the reason why this happens. So the best thing is to tell the patient not to adduct the hips or not to sit cross-legged or, you know, do any excessive ITB stretching exercises at this point. At night, the patient may be advised to sleep on the contralateral side with pillow in between the knees or by the side, if they just need to go in and roll their hip. We do not want the affected hip to dip down and thus creating that adduction at the hip, even when they're sleeping.

Surgical Intervention[edit | edit source]

Goal: Pain reduction

Recalcitrant cases with imaging concerning for abductor tears or intraarticular pathology that have not responded to extensive conservative management are often referred for surgical intervention.

  • Open or Arthroscopic Surgical Management for Recalcitrant Trochanteric Bursitis (trochanteric bursectomy).[19]



Resources
[edit | edit source]

Walker P, Ellis E, Scofield J, Kongchum T, Sherman WF, Kaye AD. Snapping Hip Syndrome: A Comprehensive Update. Orthopaedic Reviews. 2021;13(2).

References[edit | edit source]

  1. 1.0 1.1 1.2 1.3 Randelli F, Mazzoleni MG, Fioruzzi A, Giai Via A, Calvisi V, Ayeni OR. Surgical interventions for external snapping hip syndrome. Knee Surg Sports Traumatol Arthrosc. 2021 Aug;29(8):2386-2393.
  2. 2.00 2.01 2.02 2.03 2.04 2.05 2.06 2.07 2.08 2.09 2.10 2.11 Yen YM, Lewis CL, Kim YJ. Understanding and Treating the Snapping Hip. Sports Med Arthrosc Rev. 2015 Dec;23(4):194-9.
  3. 3.0 3.1 Idjadi J, Meislin R. Symptomatic snapping hip: targeted treatment for maximum pain relief. The Physician and Sportsmedicine 2004;32(1):25-31.
  4. Byrd JT. Snapping hip. Operative Techniques in Sports Medicine 2005;13(1):46-54.
  5. Baker RL, Fredericson M. Iliotibial Band Syndrome in Runners: Biomechanical Implications and Exercise Interventions. Physical médicine and réhabilitation clinics of North America, 2016; 27(1):53-77
  6. Weerakkody, Y., Bell, D. Hip bursae. Reference article, Radiopaedia.org. (accessed on 13 Mar 2022) https://doi.org/10.53347/rID-64260
  7. Bordoni B, Varacallo M. Anatomy, Bony Pelvis and Lower Limb, Iliopsoas Muscle. [Updated 2021 Jul 21]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2022 Jan-. Available from: https://www.ncbi.nlm.nih.gov/books/NBK531508/
  8. 8.0 8.1 8.2 Walker P, Ellis E, Scofield J, Kongchum T, Sherman WF, Kaye AD. Snapping Hip Syndrome: A Comprehensive Update. Orthopaedic Reviews. 2021;13(2).
  9. The Physio Channel. How to test for a Snapping Clicking Hip. 2018.Available from: https://www.youtube.com/watch?v=xkEq7nGUA1o [last accessed 13/03/2022]
  10. Sports Injuries And Rehabilitation. Ober`s Test - Hip Special Test. 2017.Available from: https://www.youtube.com/watch?v=6FEvmhjMVNs[last accessed 13/03/2022]
  11. John Gibbons. THE PSOAS Muscle - Simple test for tight HIP FLEXORS (Modified Thomas test). 2018. Available from: https://www.youtube.com/watch?v=qsgrMVuataI [last accessed 13/03/2022]
  12. Winston P, Awan R, Cassicy JD et al. Clinical examination and ultrasound of self-reported snapping hip syndrome in elite ballet dancers. Am J Sports Med. 2007;35:118-126.
  13. Spina AA. External coxa saltans (snapping hip) treated with Active Release Techniques: a case report. J Can Chiropr Assoc. 2007 Mar;51(1):23-9.
  14. Frizziero A, Vittadini F, Pignataro A, Gasparre G, Biz C, Ruggieri P, Masiero S. Conservative management of tendinopathies around hip. Muscles Ligaments Tendons J. 2016 Dec 21;6(3):281-292.
  15. Pierce TP, Kurowicki J, Issa K, Festa A, Scillia AJ, McInerney VK. External snapping hip: a systematic review of outcomes following surgical intervention: External snapping hip systematic review. Hip Int. 2018 Sep;28(5):468-472.
  16. Malinowski K, Kalinowski Ł, Góralczyk A, Ribas M, Lund B, Hermanowicz K. External Snapping Hip Syndrome Endoscopic Treatment: “Fan-like” Technique as a Stepwise, Tailor-made Solution.Arthroscopy Techniques, 2020; 9(10): e1553-e1557.
  17. Maldonado DR, Lall AC, Battaglia MR, Laseter JR, Chen JW, Domb BG. Arthroscopic Iliopsoas Fractional Lengthening. JBJS Essent Surg Tech. 2018 Nov 28;8(4):e30.
  18. Seidman AJ, Varacallo M. Trochanteric bursitis. StatPearls [Internet]. 2020 Jun 29.
  19. 19.0 19.1 19.2 Crutchfield CR, Padaki AS, Holuba KS, Arney MM, O'Connor MJ, Menge TJ, Lynch TS. Open Versus Arthroscopic Surgical Management for Recalcitrant Trochanteric Bursitis: A Systematic Review. Iowa Orthop J. 2021 Dec;41(2):45-57.
  20. 20.00 20.01 20.02 20.03 20.04 20.05 20.06 20.07 20.08 20.09 20.10 Pandya R. Snapping Hip and Trochanteric Bursitis. Physiopedia Course, 2022.
  21. 21.0 21.1 Mercadante JR, Marappa-Ganeshan R. Anatomy, Skin Bursa. [Updated 2021 Jul 27]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2022 Jan-. Available from: https://www.ncbi.nlm.nih.gov/books/NBK554438/
  22. 22.0 22.1 22.2 22.3 Schapira D, Nahir M, Scharf Y. Trochanteric bursitis: a common clinical problem. Arch Phys Med Rehabil. 1986 Nov;67(11):815-7.
  23. Fearon A, Stephens S, Cook J, Smith P, Neeman T, Cormick W, Scarvell J. The relationship of femoral neck-shaft angle and adiposity to greater trochanteric pain syndrome in women. A case-control morphology and anthropometric study. Br J Sports Med. 2012 Sep;46(12):888-92.
  24. 24.0 24.1 24.2 Mellor R, Bennell K, Grimaldi A, Nicolson P, Kasza J, Hodges P, Wajswelner H, Vicenzino B., 2018. Education plus exercise versus cortico- steroid injection use versus a wait and see approach on global outcome and pain from gluteal tendinopathy: prospective, single-blinded, randomised clinical trial. BMJ, 2018;2(361):k1662.
  25. Ganderton C, Semciw A, Cook J, Pizzari T. Does menopausal hormone therapy (MHT), exercise or a combination of both, improve pain and function in postmenopausal women with greater trochanteric pain syndrome (GTPS)? A randomised controlled trial. BMC Women's Health. 2016 Jun 16;16:32.
  26. Ho GW, Howard TM. Greater trochanteric pain syndrome: more than bursitis and iliotibial tract friction. Curr Sports Med Rep. 2012 Sep-Oct;11(5):232-8.
  27. Rothschild B. Trochanteric area pain, the result of a quartet of bursal inflammation. World J Orthop. 2013 Jul 18;4(3):100-2.
  28. 28.0 28.1 Grumet RC, Frank RM, Slabaugh MA, Virkus WW, Bush-Joseph CA, Nho SJ. Lateral hip pain in an athletic population: differential diagnosis and treatment options. Sports Health. 2010 May;2(3):191-6.
  29. kenzothiazepine. Trendelenburg Gait. 2012. Available from: https://www.youtube.com/watch?v=ZUPQp5oxXj8 [last accessed 15/03/2022]
  30. Woodley SJ, Nicholson HD, Livingstone V, Doyle TC, Meikle GR, Macintosh JE, Mercer SR. Lateral hip pain: findings from magnetic resonance imaging and clinical examination. journal of orthopaedic & sports physical therapy. 2008 Jun;38(6):313-28.
  31. FFITT Health Global. FABER Test. 2010.Available from: https://www.youtube.com/watch?v=XCOD1dkcVk0&t=43s [last accessed 15/03/2022]
  32. AIF Education. Functional Movement Test - Step Up and Down. 2013.Available from: https://www.youtube.com/watch?v=uUUDSxcO5VY[last accessed 15/03/2022]