Physical Activity and Cardiovascular Disease: Difference between revisions

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== Physical Activity Effects on Cardiovascular Diseases<br>  ==
== Physical Activity Effects on Cardiovascular Diseases<br>  ==


<br>The benefits of exercise with 1000kcal per week in secondary prevention decreases the all cause mortality around 20-30% (2) Physical activity improves systolic blood pressure, angina symptoms and exercise tolerance in patients without revascularization (3). For patients with revascularization physical activity improves quality of lives and exercise tolerance, as well as 29% of cardiac events and around 20% lower re-admission rates (4)
<br>The benefits of exercise with 1000kcal per week in secondary prevention decreases the all cause mortality around 20-30%&nbsp;<ref name="Lee I-M, Skerett PJ:">: Physical activity and all-cause mortality—What is the dose response relation? Med. Sci Sports exerc33(6Suppl):S459,2001.</ref> Physical activity improves systolic blood pressure, angina symptoms and exercise tolerance in patients without revascularization <ref name=") Hambrecht R. Wolf A, Gielen S, et al:">Effects of exercise on coronary endoothelial function in patients wwith coronary artery disease. Am J Cadriol 90:124, 2002.</ref>. For patients with revascularization physical activity improves quality of lives and exercise tolerance, as well as 29% of cardiac events and around 20% lower re-admission rates<ref name="Belardinelli R, Paolini I, Cianci G, et al:">Exercise training intervention after coronary angioplasty: The ETICA trial. J Am Coll Cardiol 37:1891, 2001.</ref>.


== Symptoms of Cardiovascular Events cited from (5)  ==
== Symptoms of Cardiovascular Events cited from (5)  ==

Revision as of 11:44, 14 March 2017

Definition (cited from WHO) [1]
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Cardiovascular diseases are a group of disorders of the heart and blood vessels and include:

• coronary heart disease: disease of the blood vessels supplying the heart muscle;

• cerebrovascular disease: disease of the blood vessels supplying the brain;

• peripheral arterial disease: disease of blood vessels supplying the arms and legs;

• rheumatic heart disease: damage to the heart muscle and heart valves from rheumatic fever, caused by streptococcal bacteria;

• congenital heart disease: malformations of heart structure existing at birth;

• deep vein thrombosis and pulmonary embolism: blood clots in the leg veins, which can dislodge and move to the heart and lungs.

Heart attacks and strokes are usually acute events and are mainly caused by a blockage that prevents blood from flowing to the heart or brain. The most common reason is a build-up of fatty deposits on the inner walls of the blood vessels. Strokes can be caused by bleeding from a blood vessel in the brain or by blood clots.

Physical Activity Effects on Cardiovascular Diseases
[edit | edit source]


The benefits of exercise with 1000kcal per week in secondary prevention decreases the all cause mortality around 20-30% [2] Physical activity improves systolic blood pressure, angina symptoms and exercise tolerance in patients without revascularization [3]. For patients with revascularization physical activity improves quality of lives and exercise tolerance, as well as 29% of cardiac events and around 20% lower re-admission rates[4].

Symptoms of Cardiovascular Events cited from (5)[edit | edit source]

Chest discomfort (pressure, squeezing, fullness, pain) Discomfort in one or both arms, back, jaw or stomach Shortness of breath Cold sweat, nausea, lightheadness

References[edit | edit source]

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  1. . http://www.euro.who.int/en/health-topics/noncommunicable-diseases/cardiovascular-diseases/cardiovascular-diseases2/definition-of-cardiovascular-diseases [Accessed 2/25/2017].
  2. : Physical activity and all-cause mortality—What is the dose response relation? Med. Sci Sports exerc33(6Suppl):S459,2001.
  3. Effects of exercise on coronary endoothelial function in patients wwith coronary artery disease. Am J Cadriol 90:124, 2002.
  4. Exercise training intervention after coronary angioplasty: The ETICA trial. J Am Coll Cardiol 37:1891, 2001.