Hypertonicity vs spasticity: Difference between revisions

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== Hypertonicity or spastic dystonia  ==
== Hypertonicity or spastic dystonia  ==


Hypertonicity or spastic dystonia is a continues increase in the muscle tension without regarding to the movement and is dependent upon afferent information from feedback following movements of stretch , decorticat and decerbrate rigidity are a form of spastic dystonia, spastic dystonia is considered to be a form of sustained efferent muscular hyperactivity , dependent on continues supraspinal derive to the alpha motor neuron&nbsp;<ref>Clinical evaluation and management of spasticity,  Jeffery et. al.2002</ref>  
Hypertonicity or spastic dystonia is a continuous increase in the muscle tension without regard to the movement being performed. It is dependent upon afferent information from feedback following movements of stretch. Decorticate and decerbrate rigidity are a form of spastic dystonia. Spastic dystonia is considered to be a form of sustained efferent muscular hyperactivity, dependent on continuous supraspinal drive to the alpha motor neuron. <ref>Clinical evaluation and management of spasticity,  Jeffery et. al.2002</ref>  


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[[Image:Decorticate.jpg]]<br>  
[[Image:Decorticate.jpg|centre]]<br>  


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== Spasticity  ==
== Spasticity  ==


*spasticity is a velocity dependent increase in muscle tone in regarding to passive movement&nbsp;
Spasticity is a velocity-dependent increase in muscle tone in response to passive movement. Pyramidal tract injury doesn't give rise to spasticity. Rather, the main symptoms are weakness and loss of dexterity which is greater in distal than in the proximal muscles. The pyramidal tract is the system which balances muscle tone.
*pyramidal&nbsp;tract lesions
*pyramidal tract injury doesn't give raise to spasticity the main symptoms are weakness and loss of dexterity which is greater in distal than in the proximal muscles  
*pyramidal tract is the system which balance the muscle tone.


=== neural component of spasticity:&nbsp;  ===
=== Neural Component of Spasticity ===


#Dorsoreticulospinal tract (DRT)&nbsp;: Has inhibitory effect on MRT &amp;VST  
#Dorsoreticulospinal tract (DRT) - has inhibitory effect on MRT and VST  
#medial reticulospinal tract (MRT) &amp;
#medial reticulospinal tract (MRT) and [[Vestibulospinal Tract|Vestibulospinal tract]] (VST): - have facilitatory effect on the extensor tone.
#[[Vestibulospinal Tract|Vestibulospinal tract]] ( VST ):<br>- they have facilitatory effect on the extensor tone<br>- all the three systems are though to inhibit flexor reflex afferents responsible for flexor spasm
 
All three systems are thought to inhibit flexor reflex afferents responsible for flexor spasm.


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== References  ==
== References  ==
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<references />  
<references />  


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[[Category:Cerebral_Palsy]]
[[Category:Cerebral_Palsy]]

Revision as of 20:48, 29 August 2016

Hypertonicity or spastic dystonia[edit | edit source]

Hypertonicity or spastic dystonia is a continuous increase in the muscle tension without regard to the movement being performed. It is dependent upon afferent information from feedback following movements of stretch. Decorticate and decerbrate rigidity are a form of spastic dystonia. Spastic dystonia is considered to be a form of sustained efferent muscular hyperactivity, dependent on continuous supraspinal drive to the alpha motor neuron. [1]


Decorticate.jpg



[edit | edit source]

Spasticity[edit | edit source]

Spasticity is a velocity-dependent increase in muscle tone in response to passive movement. Pyramidal tract injury doesn't give rise to spasticity. Rather, the main symptoms are weakness and loss of dexterity which is greater in distal than in the proximal muscles. The pyramidal tract is the system which balances muscle tone.

Neural Component of Spasticity[edit | edit source]

  1. Dorsoreticulospinal tract (DRT) - has inhibitory effect on MRT and VST
  2. medial reticulospinal tract (MRT) and Vestibulospinal tract (VST): - have facilitatory effect on the extensor tone.

All three systems are thought to inhibit flexor reflex afferents responsible for flexor spasm.


References[edit | edit source]

  1. Clinical evaluation and management of spasticity, Jeffery et. al.2002