Double Crush Syndrome: Difference between revisions
Venus Pagare (talk | contribs) No edit summary |
Venus Pagare (talk | contribs) No edit summary |
||
| Line 7: | Line 7: | ||
<br> | <br> | ||
== Hypothesis == | == Hypothesis == | ||
*It is suggested that compression of an axon at one location makes it more sensitive to effects of compression in another location,because of impaired axoplasmic flow. | |||
*Hypothetically, two lesions with little or no independent clinical ramifications, when combined, lead to appearance or magnification of symptoms | |||
*Two areas of compression affecting the same axons do not, alone, meet the criteria of the hypothesis. | |||
*By definition, a first lesion must render axons more susceptible to effects of a second, leading to more than just the combined, independent effects of two lesions | |||
<br> | |||
== References == | == References == | ||
Revision as of 13:44, 1 May 2014
Introduction[edit | edit source]
- The double crush hypothesis was first formulated in 1973 and states that axons that have been compressed at one site become especially susceptible to damage at another site.
- This theory was originally described by Upton (1973) in a study of 115 patients.
- The existence of double crush syndrome was further substantiated by Massey's (1981) study of nineteen cases of carpal tunnel syndrome co-existing with a cervical radiculopathy.
Hypothesis[edit | edit source]
- It is suggested that compression of an axon at one location makes it more sensitive to effects of compression in another location,because of impaired axoplasmic flow.
- Hypothetically, two lesions with little or no independent clinical ramifications, when combined, lead to appearance or magnification of symptoms
- Two areas of compression affecting the same axons do not, alone, meet the criteria of the hypothesis.
- By definition, a first lesion must render axons more susceptible to effects of a second, leading to more than just the combined, independent effects of two lesions
