Double Crush Syndrome: Difference between revisions
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*The existence of double crush syndrome was further substantiated by Massey's (1981) study of nineteen cases of carpal tunnel syndrome co-existing with a cervical radiculopathy. | *The existence of double crush syndrome was further substantiated by Massey's (1981) study of nineteen cases of carpal tunnel syndrome co-existing with a cervical radiculopathy. | ||
<br> | <br> | ||
== | == Hypothesis == | ||
*It is suggested that compression of an axon at one location makes it more sensitive to effects of compression in another location,because of impaired axoplasmic flow. | |||
*Hypothetically, two lesions with little or no independent clinical ramifications, when combined, lead to appearance or magnification of symptoms | |||
== References == | |||
<references /> | |||
[[Category:Musculoskeletal/Orthopaedics|Orthopaedics]] [[Category:Conditions]] | [[Category:Musculoskeletal/Orthopaedics|Orthopaedics]] [[Category:Conditions]] | ||
Revision as of 13:42, 1 May 2014
Introduction[edit | edit source]
- The double crush hypothesis was first formulated in 1973 and states that axons that have been compressed at one site become especially susceptible to damage at another site.
- This theory was originally described by Upton (1973) in a study of 115 patients.
- The existence of double crush syndrome was further substantiated by Massey's (1981) study of nineteen cases of carpal tunnel syndrome co-existing with a cervical radiculopathy.
Hypothesis[edit | edit source]
- It is suggested that compression of an axon at one location makes it more sensitive to effects of compression in another location,because of impaired axoplasmic flow.
- Hypothetically, two lesions with little or no independent clinical ramifications, when combined, lead to appearance or magnification of symptoms
