Burn Wound Assessment

Original Editor - Carin Hunter based on the course by Diane Merwarth
Top Contributors - Carin Hunter, Stacy Schiurring and Jess Bell

Important Terminology/Tissue Types[edit | edit source]

Dry Eschar: Eschar refers to the nonviable layers of skin or tissue indicating deep partial or full thickness injury. It is black, thick and leathery in appearance. This word is not synonymous with the word "scab".

Slough: Also known as fibrotic tissue or necrotic tissue. Slough is necrotic nonviable tissue found on the surface of a wound which must be removed via debridement in order for wound healing to occur. This tissue is wet, stingy, yellow or brown in colour. If autolytic or enzymatic debridement is utilised, it is easy to confuse liquifying slough with purulent drainage. Purulent drainage is a sign of infection, while liquifying slough is not.

Scab: Dry, crusty residue accumulated on top of a wound, resulting from coagulation of blood, purulent drainage, serum or a combination of all.

Pseudo-Eschar: A thick gelatinous yellow or tan film that forms with antibiotic creams mixing with wound exudate. It can often be mistaken for eschar, but it can be removed fairly easily with mechanical debridement.

Granulation Tissue: Granulation tissue is new connective tissue and microscopic blood vessels which form on the wound surface during the healing process. This tissue is bright red and has the appearance of ground meat. It is highly vascularised and will bleed easily. When granulation tissue grown past the height of and beyond the borders of the wound bed it is known as hypergranulation or over granulation tissue. Hypergranulation tissue must be removed in order for a wound to close by re-epithelisation.

Classification by Depth[edit | edit source]

Type Layers Involved Signs and Symptoms Healing Time Prognosis and Complications
First-Degree / Superficial Epidermis Red

Dry

Pain

No blisters

Re-epithelializes takes 2-5 days Heals well.

Repeated sunburns increase the risk of skin cancer later in life.

Second-Degree / Superficial Partial Thickness Epidermis and can extend into the superficial dermis Redness with a clear blister.

Blanches with pressure, but shows rapid capillary refill when released.

Generally moist

Very painful

Hair attachments are intact

Wound bed pink to red

Re-epithelializes takes 1-2 weeks Low risk of infection unless patient is compromised

No scarring typically

Oedema is common

Second-Degree / Deep Partial Thickness Extends into deep (reticular) dermis

Often causes damage to the hair follicle and glandular tissue

Appears yellow or white.

Less blanching than superficial. Sluggish capillary refill indicates vascular damage

Hair attachments are intact

Very painful to pressure and uncomfortable

Blisters are uncommon

Often moist and waxy

Wound bed shades of red, yellow, white

Re-epithelialize 2-5 weeks.

Some require surgical closure

Scarring, contractures (may require excision and skin grafting)

Oedema

Circumferential burns at risk for compartment syndrome

Increased risk of infection due to depth and impaired blood flow

Third-Degree / Full Thickness Extends through entire dermis and can often affect the underlying subcutaneous tissue Shades of brown, tan, waxy white, cherry red, sometimes with petechiae

Appearance can vary from waxy white, leathery grey or charred black.

Skin is dry, lacking in elasticity

No blanching

Not painful (nerve ending damage is common)

Stiff and white/brown. Initially painfree

Hair attachments absent

No blanch response indicates capillary destruction

Prolonged (months) and unfinished/incomplete Increased risk of infection due to capillary destruction

Eschar, or the dead, denatured skin, is removed

Results in scarring, contractures and amputation (early excision recommended)

Fourth-Degree/

Subcutaneous

Destruction of dermis and hypdermis, and into underlying fat, muscle and bone Charred with eschar

Dry

No elasticity

Initially painfree

Hair attachments absent

No blanch response indicates capillary destruction

Does not heal on its own

Requires surgery and reconstruction

Amputation

Significant functional impairment

Death

Jacksons’ Burn Wound Model[edit | edit source]

Jacksons’ Burn Wound Model[1] is a model used to understand the pathophysiology of a burn would. This model divides the wound into three zones.

  • Zone of Coagulation: This is the area central to the injury and is the area that experiences the greatest tissue damage. Often characterised by complete destruction of the capillaries leading to complete cell death. This is irreversible as there is no capillary refill.
  • Zone of Stasis or Zone of Ischaemia: This area is adjacent to the zone of coagulation and as the name suggests, it is a zone in which the there is slowing of circulating blood due to the damage. These are areas of deep partial thickness burns, or burns of indeterminate depth. This zone can usually be saved with the correct wound care. Capillaries are often compromised by oedema due to hypovolemia vasoconstrictive mediators responding to injury. It is reversible if capillary flow can be restored.
  • Zone of Hyperemia: This zone is circumferential and is characterised by the eased blood supply and inflammatory vasodilation. This tissue has a good recovery rate, as long as there are no complications, such as severe sepsis or prolonged hypo-perfusion. increased blood flow due to release of histamine.  This area will recover completely without intervention unless complications occur, characterised by superficial and superficial partial thickness burns and has a robust capillary refill.

Burn Wound Conversion[edit | edit source]

Burn Wound Conversion: [2] This refers to the worsening of tissue damage in a superficial burn which previously was expected to spontaneously heal, but it increases in tissue depth into a deeper wound which requires excision.

Potential Causes: 

  • Dessication
  • Infection
  • Oedema

Total Body Surface Area[edit | edit source]

The Parkland Burn Formula is the most widely used formula to estimate the fluid resuscitation required by a burns patient on hospital admission, usually within the first 24 hours. When applying this formula, the first step is to calculate the percentage of body surface area (BSA) damaged, which is most commonly done by the "Wallace Rule of Nines".[3] When conducting a paediatric assessment, the Lund-Browder Method is commonly used, as children have a greater percentage surface area of their head and neck compared to an adult. The formula recommends 4 millilitres per kilogram of body weight in adults (3 millilitres per kilogram in children) per percentage burn of total body surface area (%TBSA) of crystalloid solution over the first 24 hours of care.[4]

4 mL/kg/%TBSA (3 mL/kg/%TBSA in children) = total amount of crystalloid fluid during first 24 hours

The latest research has indicated that while this method is still in use, the fluid levels should be constantly monitored, while assessing the urine output,[5] to prevent over-resuscitation or under-resuscitation.[6]

Calculation of Percentage Burn of Total Body Surface Area[edit | edit source]

  1. The Rule of Nine
  2. Lund-Browder Method
  3. Palmer Method
1. The Rule of Nine[edit | edit source]
Body Part Percentage for Rule of Nine
Head and Neck 9%
Entire chest 9%
Entire abdomen 9%
Entire back 18%
Lower Extremity 18% each
Upper Extremity 9% each
Groin 1%
2. Lund-Browder Method[edit | edit source]
Lund and Browder Chart.jpg
3. Palmar Surface Method[edit | edit source]

The "Rule of Palm" or Palmar Surface Method can be used to estimate body surface area of a burn. This rule indicates that the palm of the patient, with the exclusion of the fingers and wrist, is approximately 1% of the patients body surface area. When a quick estimate is required, the percentage body surface area will be the number of the patient's own palm it would take to cover their injury. It is important to use the patient's palm and not the provider's palm.

References[edit | edit source]

  1. Harish V, Li Z, Maitz PK. First aid is associated with improved outcomes in large body surface area burns. Burns. 2019 Dec 1;45(8):1743-8.
  2. Palackic A, Jay JW, Duggan RP, Branski LK, Wolf SE, Ansari N, El Ayadi A. Therapeutic Strategies to Reduce Burn Wound Conversion. Medicina. 2022 Jul;58(7):922.
  3. Bereda G. Burn Classifications with Its Treatment and Parkland Formula Fluid Resuscitation for Burn Management: Perspectives. Clinical Medicine And Health Research Journal. 2022 May 12;2(3):136-41.
  4. Mehta M, Tudor GJ. Parkland formula. 2019
  5. Ahmed FE, Sayed AG, Gad AM, Saleh DM, Elbadawy AM. A Model for Validation of Parkland Formula for Resuscitation of Major Burn in Pediatrics. The Egyptian Journal of Plastic and Reconstructive Surgery. 2022 Apr 1;46(2):155-8.
  6. Ete G, Chaturvedi G, Barreto E, Paul M K. Effectiveness of Parkland formula in the estimation of resuscitation fluid volume in adult thermal burns. Chinese Journal of Traumatology. 2019 Apr 1;22(02):113-6.