Whiplash Associated Disorders

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Clinically Relevant Anatomy
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Whiplash and whiplash associated disorders (WAD) affects variable areas of the cervical spine, depending on the force and direction of impact as well as many other factors. In a whiplash injury, bony structures, ligamentous structures, muscles, neurological structures, and other connective tissue may be affected. Anatomic causes of pain can be any of these structures, with the strain injury resulting in secondary edema, hemorrhage, and inflammation.

Mechanism of Injury / Pathological Process
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The mechanism of injury is variable, usually involving a motor vehicle accident but also including causes such as sports injury, child abuse, blows to the head from a falling object, or similar accelleration-decceleration event.

Clinical Presentation[edit | edit source]

The most common presentation will be sub-occipital headaches and/or neck pain that is constant or motion-induced. There may be up to 48 hrs delay of symptom onset from the initial injury. Other signs include neurologic signs, dizziness, tinnitus, visual disturbances, UE radicular pain, difficulty sleeping due to pain, and difficulty concentrating/poor memory. (eMedicine) It is important to provide a thorough spinal exam and neurologic exam in patient with WAD to screen for delayed-onset of cervical spine instability or myelopathy. [1]

QTFC (Quebec Task Force Classification)
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QTFC Grade
Clinical presentation
0

No complaint about neck pain

No physical signs

I

Nec complaints of pain, stiffness or tenderness only

No physical signs

II

Neck complaint

Musculoskeletal signs including

  • decreased ROM
  • point tenderness
III

Neck complaint

Musculosceletal signs

Neurological signs including:

  • decreased or absent deep tendon reflexes
  • muscle weakness
  • sensory deficits
IV
Neck complaint and fracture or dislocation



MQTFC (Modified Quebec Task Force Classification) [2]



Proposed

classification grade

Physical and psychological impairments present
WAD 0

No complaints about neck pain

No physical signs

WAD I

No complaints of pain, stiffness or tenderness only

No physical signs

WAD IIA

Neck complaint

Motor impairment

  • decreased ROM
  • altered muscle recruitment patterns (CCFT)

Sensory Impairment

  • local cervical mechanical hyperalgesia
WAD IIB

Neck complaint

Motor impairment

  • decreased ROM
  • altered muscle recruitment patterns (CCFT)

Sensory Impairment

  • local cervical mechanical hyperalgesia

Psychological impairment

  • elevated psychological distress (GHQ, TAMPA)
WAD IIC
Neck complaint

Motor impairment

  • decreased ROM
  • altered muscle recruitment patterns (CCFT)
  • increased JPE

Sensory Impairment

  • local cervical mechanical hyperalgesia
  • generalized sensory hypersensitivity (mechanical, thermal, ULNT)
  • Some may show SNS disturbances

Psychological impairment

  • elevated psychological distress (GHQ, TAMPA)
  • elevated levels of acute posttraumatic stress (IES)
WAD III

Neck complaint

Motor impairment

  • decreased ROM
  • altered muscle recruitment patterns (CCFT)
  • increased JPE

Sensory Impairment

  • local cervical mechanical hyperalgesia
  • generalized sensory hypersensitivity (mechanical, thermal, ULNT)
  • Some may show SNS disturbances

Neurological signs of conduction loss including:

  • decrease or absent deep tendon reflexes
  • muscle weakness
  • sensory deficits

Psychological impairment

  • elevated psychological distress (GHQ, TAMPA)
  • elevated levels of acute posttraumatic stress (IES)
WAD IV
Fracture or dislocation



Diagnostic Procedures[edit | edit source]

Canadian C-Spine Rule (CCR): algorithm to determine the necessity for cervical spine radiography in alert and stable patients presenting with trauma and cervical spine injury. [3]



Management / Interventions
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Management approaches for patients with WAD are poorly researched. These patients often do not fit into treatment categories as defined for other cervical pain problems due to multiple factors, and even within the WAD group there are multiple variances which warrant individualized treatment approaches. The most recent evidence supports the use of Sterling's classification system for WAD. [4] 

Differential Diagnosis
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Cervical radiculopathy
Facticious disorder
Polymyalgia Rheumatica
Traumatic Brain Injury
Cervical herniated disk
Cervical myelopathy
Cervical osteoarthritis
Infection or osteomyelitis
Inflammatory rheumatologic disease
Malingering
Psychogenic pain disorder
Referred pain from cardiothoracic structures
Tumor or malignancy of cervical spine

Vascular abnormality of cervical structures

Key Evidence[edit | edit source]

"Whiplash associated disorders- redefining whiplash and its management" by the Quebec Task Force: a critical evaluation.

Resources
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www.som.uq.edu.au/whiplash

Evidence-based summary

Case Studies[edit | edit source]

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References[edit | edit source]

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  1. Delfini R, Dorizzi A, Facchinetti G, Faccioli F, Galzio R, Vangelista T. Delayed post-traumatic cervical instability. Surg Neurol. 1999;51:588-95.
  2. Sterling M., Man Ther. 2004 May;9(2):60-70. A proposed new classification system for whiplash associated disorders--implications for assessment and management.
  3. Stiell IG, Clement CM, McKnight RD, Brison R, Schull MJ, Rowe BH, Worthington JR, Eisenhauer MA, Cass D, Greenberg G, MacPhail I, Dreyer J, Lee JS, Bandiera G, Reardon M, Holoroyd B, Lesiuk H, Wells GA. The Canadian c-spine rule versus the NEXUS low-risk criteria in patients with trauma. N Engl J Med. 2003;349(26): 2510-2518.
  4. Sterling M, Jull G, Kenardy J. Physical and psychological factors maintain long-term predictive capacity post-whiplash injury. Pain. 2006;122:102-108.