Vasovagal Syncope: Difference between revisions
No edit summary |
No edit summary |
||
| Line 5: | Line 5: | ||
</div> | </div> | ||
== Introduction == | == Introduction == | ||
Vasovagal syncope, also known as neurocardiogenic or reflex syncope, is caused by a sudden drop in blood pressure, often triggered by a reaction to something. | Vasovagal syncope (VVS), also known as neurocardiogenic or reflex syncope, is caused by a sudden drop in blood pressure, often triggered by a reaction to something. VVS is the most common form of syncope, and although usually harmless, its frequent occurrence adversely affects quality of life and increase the risk of adverse events<ref>Longo S, Legramante JM, Rizza S, Federici M. [https://www.sciencedirect.com/science/article/abs/pii/S0953620523000973 Vasovagal syncope:] An overview of pathophysiological mechanisms. European Journal of Internal Medicine. 2023 Apr 6.Available: https://www.sciencedirect.com/science/article/abs/pii/S0953620523000973<nowiki/>(accessed 4.5.2024)</ref>. Diagnosis and management of vasovagal syncope is hard as the differential diagnoses are manyfold. In most cases vasovagal syncope is treated conservatively.<ref>Jeanmonod R, Sahni D, Silberman M. [https://www.ncbi.nlm.nih.gov/books/NBK470277/ Vasovagal Episode]. StatPearls. Treasure Island (FL).Available:https://www.ncbi.nlm.nih.gov/books/NBK470277/ (accessed 3.5.2024)</ref> | ||
== | == Epidemiology == | ||
VVS, the most common form of syncope in adults, is the cause of 85% of syncopal events in people younger than 40 years. In older persons, more than half of syncopal episodes are due to VVS. VVS needs a working autonomic nervous system its occurrence is low in those with autonomic nervous system dysfunction, eg Parkinson disease. Lifetime incidence a VVS is more than 33%.<ref name=":0">Jeanmonod R, Sahni D, Silberman M. [https://www.ncbi.nlm.nih.gov/books/NBK470277/ Vasovagal Episode]. StatPearls. Treasure Island (FL).Available:https://www.ncbi.nlm.nih.gov/books/NBK470277/ (accessed 4.5.2024)</ref> | |||
== | == Management == | ||
VVS is on the whole treated conservatively. If known and avoidable, patients need to avoid triggers. Patients are may need to drink more fluids to improve their volume status and to slowly change positions. If the patient experiences "warning signs" of a VVS educate them to place themselves in a supine position if they feel an imminent event. Most patients with VVS have a history of a syncopal prodrome. Patients may describe a feeling of lightheadedness, feelings of warmth and nausea, tunnel vision, ringing in their ears, and profuse sweating. Preventative strategies at this stage will often prevent a VVS event by preventing traumatic risk from a fall. In refractory or disabling cases and cases of prolonged asystole, cardiac pacing is a therapeutic option.<ref name=":0" /> | |||
== Resources == | == Resources == | ||
Revision as of 03:51, 4 May 2024
Original Editor - User Name
Top Contributors - Lucinda hampton
Introduction[edit | edit source]
Vasovagal syncope (VVS), also known as neurocardiogenic or reflex syncope, is caused by a sudden drop in blood pressure, often triggered by a reaction to something. VVS is the most common form of syncope, and although usually harmless, its frequent occurrence adversely affects quality of life and increase the risk of adverse events[1]. Diagnosis and management of vasovagal syncope is hard as the differential diagnoses are manyfold. In most cases vasovagal syncope is treated conservatively.[2]
Epidemiology[edit | edit source]
VVS, the most common form of syncope in adults, is the cause of 85% of syncopal events in people younger than 40 years. In older persons, more than half of syncopal episodes are due to VVS. VVS needs a working autonomic nervous system its occurrence is low in those with autonomic nervous system dysfunction, eg Parkinson disease. Lifetime incidence a VVS is more than 33%.[3]
Management[edit | edit source]
VVS is on the whole treated conservatively. If known and avoidable, patients need to avoid triggers. Patients are may need to drink more fluids to improve their volume status and to slowly change positions. If the patient experiences "warning signs" of a VVS educate them to place themselves in a supine position if they feel an imminent event. Most patients with VVS have a history of a syncopal prodrome. Patients may describe a feeling of lightheadedness, feelings of warmth and nausea, tunnel vision, ringing in their ears, and profuse sweating. Preventative strategies at this stage will often prevent a VVS event by preventing traumatic risk from a fall. In refractory or disabling cases and cases of prolonged asystole, cardiac pacing is a therapeutic option.[3]
Resources[edit | edit source]
- bulleted list
- x
or
- numbered list
- x
References[edit | edit source]
- ↑ Longo S, Legramante JM, Rizza S, Federici M. Vasovagal syncope: An overview of pathophysiological mechanisms. European Journal of Internal Medicine. 2023 Apr 6.Available: https://www.sciencedirect.com/science/article/abs/pii/S0953620523000973(accessed 4.5.2024)
- ↑ Jeanmonod R, Sahni D, Silberman M. Vasovagal Episode. StatPearls. Treasure Island (FL).Available:https://www.ncbi.nlm.nih.gov/books/NBK470277/ (accessed 3.5.2024)
- ↑ 3.0 3.1 Jeanmonod R, Sahni D, Silberman M. Vasovagal Episode. StatPearls. Treasure Island (FL).Available:https://www.ncbi.nlm.nih.gov/books/NBK470277/ (accessed 4.5.2024)
