Assessment of Tennis Elbow
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Top Contributors - Mariam Hashem, Kim Jackson, Jess Bell, Nupur Smit Shah, Tony Lowe, Tarina van der Stockt, Rachael Lowe, Simisola Ajeyalemi, Lucinda hampton, Olajumoke Ogunleye and Robin Tacchetti
Introduction[edit | edit source]
Tennis Elbow, also known as Lateral Epicondylitis, is described as pain over the lateral epicondyle of the humerus. It is a common presentation between 35-45 years of age. Smoking, obesity, manual work requiring repetitive loading of wrist extensors and tennis players are considered to be risk factors of Tennis Elbow[1].
Tennis Elbow has great effects on the quality of life as well as the participation in work, sports and leisure activities.
Lateral Tendinopathy is seen in 1-3% of the general population
Although up to 90% of the presentations are self-limiting,, not all of them experience full recovery and the pain and discomfort last up to a year. Recurrence is also common in Tennis Elbow and around 5% need surgery
Pathophysiology[edit | edit source]
The structural pathology is not always evident in various clinical presentation. In fact, some studies reported discordance between clinical severity and tendon pathology in patients with tendinopathy. This drives our attention to the multi-factorial nature of many MSK conditions. Psychological factors, central sensitization and/or other CNS-mediated factors may play roles in the onset and prognosis of the condition[1].
From a histological point of view, Lateral Teninopathy seems to be progressing as any other tendinopathy, increased cellularity, an accumulation of ground substance, collagen disorganization, and neurovascular ingrowth. In the case of Tennis Elbow this process is observed in the deep and anterior fibers of the extensor carpi radialis brevis (ECRB). In sever presentations, the ECRB is often merged with the lateral collateral ligament (LCL), which fuses with the annular ligament of the proximal radioulnar joint.
