Arthrogenic Muscle Inhibition: Difference between revisions

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== Mechanism ==
== Mechanism ==
Change in afferent input to the spinal cord from the joint mechanoreceptors (Ruffini endings, Pacinian capsule, Golgi tendon organ, Free nerve endings) appears to play a strong role in the mechanism of AMI. Following an injury, the joint mechanoreceptors activation act on inhibitory interneurons synapsing on the motoneuron pool of joint musculature, thus, the contractions created by the motoneuron pool decrease. <ref name=":0" />
Change in afferent input to the spinal cord from the joint mechanoreceptors (Ruffini endings, Pacinian capsule, Golgi tendon organ, Free nerve endings) appears to play a strong role in the mechanism of AMI. Following an injury, the joint mechanoreceptors activation acts on inhibitory interneurons synapsing on the motoneuron pool of joint musculature, thus, the contractions created by the motoneuron pool decrease. <ref name=":0" />


== Clinical Presentation  ==
== Clinical Presentation  ==
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== Diagnostic Procedures  ==  
== Diagnostic Procedures  ==  


== Outcome Measures  ==  
== Outcome Measures  ==
 
* Voluntary Force Measurement
* The Hoffmann Reflex


== Management / Interventions ==
== Management / Interventions ==
Exercise
Cryotherapy
Neuromuscular electrical stimulation
Transcutaneous electrical nerve stimulation
Vibration


Ultrasound
* Pharmacological Agents
* Exercise
* Cryotherapy
* Neuromuscular electrical stimulation
* Transcutaneous electrical nerve stimulation
* Vibration
* Ultrasound


== Differential Diagnosis<br>  ==  
== Differential Diagnosis<br>  ==  

Revision as of 15:38, 10 March 2024

Original Editor - Sehriban Özmen
Top Contributors - Sehriban Ozmen

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Definition[edit | edit source]

Arthrogenic muscle inhibition (AMI) is the presynaptic, ongoing reflex inhibition of the musculature surrounding the injured joint. It is a natural response generated by the human body to protect the joint from further damage. [1]

Mechanism[edit | edit source]

Change in afferent input to the spinal cord from the joint mechanoreceptors (Ruffini endings, Pacinian capsule, Golgi tendon organ, Free nerve endings) appears to play a strong role in the mechanism of AMI. Following an injury, the joint mechanoreceptors activation acts on inhibitory interneurons synapsing on the motoneuron pool of joint musculature, thus, the contractions created by the motoneuron pool decrease. [1]

Clinical Presentation[edit | edit source]

Quadriceps weakness following Anterior Cruciate Ligament Injury:

Diagnostic Procedures[edit | edit source]

Outcome Measures[edit | edit source]

  • Voluntary Force Measurement
  • The Hoffmann Reflex

Management / Interventions[edit | edit source]

  • Pharmacological Agents
  • Exercise
  • Cryotherapy
  • Neuromuscular electrical stimulation
  • Transcutaneous electrical nerve stimulation
  • Vibration
  • Ultrasound

Differential Diagnosis
[edit | edit source]

Resources
[edit | edit source]

References[edit | edit source]

  1. 1.0 1.1 Hopkins JT, Ingersoll CD. Arthrogenic muscle inhibition: a limiting factor in joint rehabilitation. Journal of sport rehabilitation. 2000 May 1;9(2):135-59.