Evidence Based Assessment of Pain in Displaced Persons - Case Study 1: Difference between revisions

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== Introduction ==
== Introduction ==
Mr. A. was referred to physiotherapy for pain and severe limitation at his right shoulder. He was detained in his origin country 5 years ago and subjected to ill-treatment during that period. He was beaten and pushed down from the stairs, fell on his shoulders several times. During the following months after detention he couldn’t receive any treatment for his shoulder and the physical complaints have became worse from day to day. He has became more and more disabled because of his shoulder pain.
Mr. S. was referred to physiotherapy for debilitating pain at his left calf, ankle and foot. His complaints started 4 years ago after he was shot from his hip. The bullet followed a diagonal track and fragmentized in his lower abdomen. He was operated in a military hospital and put into prison where he was subjected to torture after his medical treatment. The pain below his knee started those days and worsened gradually until today. The pain had constant sharp, burning character and increased with movement and standing. He was also suffering from regular face pain and headaches.


He stated that he was diagnosed with gastritis, hypertension and diabetes and he is slightly overweight. He also stated that he is suffering from sleep disturbance since he feels too much pain when he lies on his shoulder.
He didn’t have any communicable or non-communicable disease but was suffering from insomnia and PTSD symptoms as well as anxiety. He had positive expectations about physiotherapy but stated that he was extremely concerned and tired because of his pain. His CSI score was above the clinical threshold.  


Physical examination revealed significant loss of active and passive range of motion and weakness at the right shoulder. He was constantly keeping his arm and shoulder in a protective posture and there was sensitivity to palpation all over the shoulder joint. Due to over reaction to palpation and manual testing, proper evaluation couldn’t be done. Active flexion at shoulder was only 40 degrees and there was severe crepitation during the movement.  
Physical examination revealed sensitivity to palpation at lower abdominal area and below the knee. Hyperalgesia and allodynia were present below the knee. He didn’t have any loss of ROM or muscle strength in both extremities but he had pale skin and hair loss at left foot and ankle. Interestingly, neural tension tests didn’t provoke his pain. Physical evaluation for tension type headache and cervicogenic headache was negative.


He generally had positive expectations from the physiotherapy process. But he was extremely concerned about moving his arm due to fear of pain. His CSI score was below the clinical threshold but he showed high scores in PSQI and TSK. MRI screening was conducted in order to understand the clinical situation better and it showed muscle-tendon tears at supraspinatus and biceps long head as well as Hill-Sachs lesion.
He was referred for advanced screening but EMG and doppler-ultrasound didn’t show any abnormality in nerve conduction or circulation. MRI screening didn’t show any neurological finding can be related with headache. But a second referral to orthopaedist revealed a fracture in his nose remained from prison days and it was the probable cause of head and face pain.
 
In the light of the information collected from the evaluation it can be said that while he had significant structural damage and related nociceptive input, his exaggerated reaction to palpation and movement was not compatible with his injuries. Seemingly the main reason behind his disability was fear of pain and movement. So it was essential to address cognitive and behavioral contributors before expecting any improvement in nociceptive contributors. In addition to that he had important comorbidities such as sleep disturbance, diabetes and hypertension which easily hinder tissue healing and decrease pain threshold. That’s why these problems were also needed to be targeted.


Based on the collected information and findings, it was difficult to establish a clear hypothesis but nervous system dysfunction appeared to be main driver of the pain. A neurovascular compromise due to gunshot wound might have caused neuropathic pain and through the time it might have triggered central sensitization with the contribution of PTSD symptoms and constant headaches.
== Resources  ==
== Resources  ==
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Introduction[edit | edit source]

Mr. S. was referred to physiotherapy for debilitating pain at his left calf, ankle and foot. His complaints started 4 years ago after he was shot from his hip. The bullet followed a diagonal track and fragmentized in his lower abdomen. He was operated in a military hospital and put into prison where he was subjected to torture after his medical treatment. The pain below his knee started those days and worsened gradually until today. The pain had constant sharp, burning character and increased with movement and standing. He was also suffering from regular face pain and headaches.

He didn’t have any communicable or non-communicable disease but was suffering from insomnia and PTSD symptoms as well as anxiety. He had positive expectations about physiotherapy but stated that he was extremely concerned and tired because of his pain. His CSI score was above the clinical threshold.

Physical examination revealed sensitivity to palpation at lower abdominal area and below the knee. Hyperalgesia and allodynia were present below the knee. He didn’t have any loss of ROM or muscle strength in both extremities but he had pale skin and hair loss at left foot and ankle. Interestingly, neural tension tests didn’t provoke his pain. Physical evaluation for tension type headache and cervicogenic headache was negative.

He was referred for advanced screening but EMG and doppler-ultrasound didn’t show any abnormality in nerve conduction or circulation. MRI screening didn’t show any neurological finding can be related with headache. But a second referral to orthopaedist revealed a fracture in his nose remained from prison days and it was the probable cause of head and face pain.

Based on the collected information and findings, it was difficult to establish a clear hypothesis but nervous system dysfunction appeared to be main driver of the pain. A neurovascular compromise due to gunshot wound might have caused neuropathic pain and through the time it might have triggered central sensitization with the contribution of PTSD symptoms and constant headaches.

Resources[edit | edit source]

References[edit | edit source]