Cubital Tunnel Syndrome

Definition/Description[edit | edit source]

Ulnar Nerve Anatomy including Cubital Tunnel

Cubital tunnel syndrome (CBTS) is a peripheral nerve compression syndrome. It is an irritation or injury of the ulnar nerve in the cubital tunnel at the elbow. This is also termed ulnar nerve entrapment and is the second most common compression neuropathy in the upper extremity after carpal tunnel syndrome.[1] [2] It represents a source of considerable discomfort and disability for the patient and may, in extreme, cases lead to a loss of function of the hand. Cubital tunnel syndrome is also often misdiagnosed.[3] 

Peripheral nerve compression syndromes are characterised by chronic irritation and pressure lesions on the sites where nerves have to pass through narrow anatomic spaces and fibro-osseous structures. The main clinical manifestation of this type of compression are paresthesia, sensory impairment and paresis. [4]

Cubital tunnel syndrome can also be caused by traction, pressure or ischemia of the ulnar nerve which passes through the cubital tunnel at the medial side of the elbow.[5] Pain or paraesthesia in the fourth and fifth finger and pain in the medial aspect of the elbow, which may extend proximally or distally, is caused by compression of the ulnar nerve. There is only limited evidence that proves the effectiveness of nonsurgical and surgical interventions to treat cubital tunnel syndrome. [3] [6]

Clinically Relevant Anatomy[edit | edit source]

Anatomy of Cubital Tunnel

Cubital tunnel syndrome is a progressive entrapment neuropathy of the ulnar nerve at the medial aspect of the elbow. The ulnar nerve, which is a motor and sensory nerve, is formed from the medial cord of the brachial plexus, which originates from nerve roots C8 and T1.[7][8][9] The ulnar nerve travels down the posterior aspect of the arm to eventually traverse posterior to the medial epicondyle through an area known as the cubital tunnel. The cubital tunnel extends from the medial epicondyle of the humerus to the olecranon process of the ulna.[10] The nerve runs superficial to the ulnar collateral ligament (UCL) and deep to the aponeurotic attachment of the flexor carpi ulnaris (FCU), which is also known as Osborne’s ligament. Once the ulnar nerve reaches the proximal border of Osborne’s ligament it is located in the cubital tunnel.

The roof of the cubital tunnel is formed by the cubital tunnel retinaculum which is about 4 mm between the medial epicondyle and the olecranon. The floor of the tunnel consists of the elbow joint capsule and the posterior band of the medial collateral ligament of the elbow. It contains several structures of which the most important is the ulnar nerve.[3]

After passing through the cubital tunnel, the ulnar nerve passes deep into the forearm between the ulnar and humeral heads of the flexor carpi ulnaris.

Ulnar nerve entrapment can occur at 5 possible sites around the elbow:

  • Arcade of Struthers (approximately 10cm proximal to the medial epicondyle)
  • Medial intermuscular septum (runs from the arcade to the epicondyle)
  • Medial epicondyle
  • Cubital tunnel (retinaculum)
  • Deep flexor pronator aponeurosis (about 5cm distal to the epicondyle)[5]

Out of all the sites, the cubital tunnel is the most common site for entrapment.[1]

Epidemiology /Etiology[edit | edit source]

Overhead throwing is a risk factor for cubital tunnel syndrome

Cubital tunnel syndrome is the second most commonly reported upper extremity entrapment neuropathy and is the most common ulnar nerve neuropathy.[11][12] Cubital tunnel syndrome may be a result of direct or indirect trauma due to the ulnar nerve's vulnerability to traction, friction and compression. Traction injuries may be the result of longstanding valgus deformity and flexion contractures, but are most common in throwing athletes due to extreme valgus stress placed on the arm. [13] One of the most common pathogenetic mechanisms is intermittent traction when the ulnar nerve becomes fixed at a single or several points which limits the free gliding of the nerve.[5] Compression of the nerve at the cubital tunnel may occur due to reactive changes at the MCL, adhesions within the tunnel, hypertrophy of the surrounding musculature, or joint changes.[14]

This syndrome can be classified into primary or secondary causes:

  • Primary (idiopathic) include anatomical variants such as subluxation of the ulnar nerve or an epitrochlearis-anconeus muscle which is a rarely seen cause of cubital tunnel syndrome. [15]
  • Secondary (symptomatic) include a delayed ulnar paresis due to trauma or elbow arthrosis. It can also be caused by extra neural or less commonly, intraneural masses, such as a lipoma or ganglion.[4] 

There are many factors that can lead to cubital tunnel syndrome. They include:

  • Mechanical factors such as stretching of, friction on or compression of the ulnar nerve [16] [4]
  • Direct trauma or other space-occupying lesions, repetitive elbow flexion/extension, repetitive overhead activities, traction, subluxation of the ulnar nerve from the ulnar groove, metabolic disorders, congenital deformities, synovial cysts, anatomical irregularities, arthritis, joint inflammation, and occupational/athletic factors.[16] 

Risk factors include:[9][17][18]

  • head injuries with upper extremity flexion contractures
  • > 40 years old
  • overhead throwing activities
  • work which involves prolonged periods of elbow flexion, such as holding a telephone
  • persistently resting elbows on a hard surface
  • obesity

Symptoms can sometimes be associated with other conditions such as, osteoarthritis, rheumatoid arthritis and other diseases, for example, diabetes mellitus and haemophilia and symptoms can be aggravated by alcoholism, obesity and smoking.

Characteristics/Clinical Presentation[edit | edit source]

  • Depending on the duration and progression of the disorder, patients will present with similar, but a specific set of symptoms (see Table 1).[11]
  • Primary symptoms are typically medial elbow pain or aching in the forearm.[19] Numbness and tingling may also be present in the 4th and 5th digits, the ulnar side of the dorsum of the hand and the hypothenar eminence.[20][21]
  • The patient may also report non-painful snapping or popping during active and passive flexion and extension of the elbow.
  • Wartenberg sign (abduction of the fifth digit due to weakness of the third palmar interosseous muscle) may be present.
  • Active and passive ROM may not be decreased.
  • The ulnar nerve may be enlarged or palpable and tender in the groove.[22]
  • On observation, there may be atrophy of the intrinsic muscles of the hand, which is often not noticed by the patient, with abnormal claw posture of the 4th and 5th fingers [4]
Table 1 - Modified from McGowan's grading system found in Coppieters et al

There are many different ways to grade this neuropathy. A number of studies have investigated whether or not these ways have clinical significance and if they can be used as guidelines for therapy. The studies are, however, inconclusive.[23] [24]
Patients suffering from cubital tunnel syndrome are 4 times more likely to present with atrophy than patients suffering from carpal tunnel syndrome. Ulnar nerve dysfunction has been divided into three categories by McGowan and further modified by Dellon:

  • Mild nerve dysfunction implies intermittent paraesthesia and subjective weakness.
  • Moderate dysfunction presents with intermittent paraesthesia and measurable weakness.
  • Severe dysfunction is characterized by persistent paraesthesia and measurable weakness.[1]

Diagnostic Procedures[edit | edit source]

Diagnosis is established through patient history and a physical examination, in conjunction with the findings of electro-physiologic studies and imaging.


  • high-resolution neuro-ultrasonography shows changes in the size and position of the ulnar nerve at the elbow (also changes in the echotexture of the nerve)
  • magnetic resonance neurography (MRN) shows structural changes of the ulnar nerve and its environment [4] 
  • X-rays can be used to look for degenerative changes of the cervical spine and elbow, as well as bony compression from spurs or previous fractures.

Electro-physiological studies:

In establishing diagnosis, neurophysiological studies are helpful and should be done if surgery is planned, in order to document preoperative baseline. Ulnar nerve velocity of <50 m/s at the elbow is considered positive for cubital tunnel syndrome. [20]
Tinel’s signs are also used in the diagnostic procedure as well as in the diagnosis of tarsal tunnel syndrome.[23]

Outcome Measures[edit | edit source]

These represent a number of outcome measures that have been used.[25] Additional measures can be, Quick Disabilities of the Arm, Shoulder and Hand questionnaire and the Short Form-12. [26]

Examination[edit | edit source]

Accurate diagnosis includes assessing the following:

  • sensory changes in the ulnar nerve distribution (½ of the 4th digit and entirety of the 5th)
  • pain
  • atrophy of the intrinsic muscles of the hand innervated by the ulnar nerve
  • neural provocation test of the ulnar nerve
  • sparing of the flexor carpi ulnaris muscle[27]

Tests used to confirm the diagnosis of cubital tunnel syndrome are those linking ulnar neuropathy and the elbow. These tests should evoke provocative signs as a reaction to confirm the syndrome, such as: elbow flexion reproducing symptoms, positive Tinel’s sign tested at the elbow or a sign of instability, for example snapping of the ulnar nerve over the medial epicondyle with elbow flexion.[5]


Elbow Flexion Test

Typically performed bilaterally with the shoulder in full external rotation and the elbow actively held in maximal flexion with wrist extension held for one minute. Symptoms are produced as maximal elbow flexion reduces the cubital tunnel volume by approximately 55% causing increased neural pressure on the ulnar nerve.[12] This test can include additional components such as wrist extension and wrist flexion or sustained maximal elbow flexion for up to 3 minutes.[12] A positive test is reproduction of pain at the medial aspect of the elbow and numbness and tingling in the ulnar distribution on the involved side. This test has a high positive predictive value (0.97), indicating a high probability of cubital tunnel syndrome if positive, with high specificity (0.99) and sensitivity (0.75).[30]

Pressure Provocative Test:

Pressure is applied to the ulnar nerve at the cubital tunnel with the UE positioned as in the elbow flexion test for 30 seconds. Sensitivity with this test is high (0.91).[31]


Tinel Sign:

Reproduction of tingling and numbness into the 4th and 5th digits by tapping of the ulnar nerve at the cubital tunnel. Test specificity is 0.98 and sensitivity is 0.70.[34] The clinician will proceed with percussions on the ulnar nerve as it passes through the cubital tunnel after the ulnar groove, posterior of the medial epicondyle of the humerus. [11][12] There is no agreed consensus on the number of percussions, but 4 to 6 taps should be sufficient to reproduce symptoms. A positive test is the reproduction of tingling and numbness in the ulnar nerve distribution on the involved side. Caution must be taken, however, when interpreting the results as a positive test has been found in 24% of asymptomatic subjects and it could also be negative for those in the advanced stage of the diagnosis due to the nerve no longer regenerating. [34]

Scratch Collapse Test:

The patient’s skin is lightly scratched over the area of nerve compression whilst resisted bilateral shoulder external rotation is performed. A brief loss of muscle resistance will be elicited if the patient has allodynia due to compression neuropathy. Sensitivity for the scratch collapse is 69% compared with 54% and 46% for the Tinel test and elbow flexion compression test, respectively. Of all tests for cubital tunnel, the Tinel test has the highest negative predictive value (98%).[1] Research suggests the Scratch collapse test has a significantly higher sensitivity than Tinel's test and the flexion/nerve compression test for carpal tunnel and cubital tunnel syndromes. This novel test provides a useful addition to existing clinical maneuvers in the diagnosis of these common nerve compression syndromes[35].

Medical Management[edit | edit source]


Operative Management:[edit | edit source]

Indications for surgical intervention include moderate muscle weakness without response to conservative treatment after 3 months and an electrodiagnostic test of less than 39-50 meters per second across the elbow.[34][37][1] Surgery may also be indicated in cases of:

  • progressive symptoms
  • sensorimotor deficits
  • lack of clinical and electro-neurographic improvement
  • worsening of the objective findings on follow up several weeks after the initial visit.

There have been several surgical techniques advocated for cubital tunnel syndrome including:[38]

Simple decompression:[edit | edit source]

A release of Osborne’s ligament through an incision traversing in a proximal to distal direction throughout the length of the ligament increasing the space in the cubital tunnel. A 6 to 10cm incision is made along the course of the ulnar nerve between the medial epicondyle and the olecranon. Care should be taken to avoid the branches of the medial antebrachial cutaneous nerve. Osbourne’s ligament is released as are the FCU superficial and deep fascia. It has been shown to be successful in treating cubital tunnel syndrome. Data suggests that in situ decompression is a reliable treatment with a low failure rate and anterior trans-positioning can be used to treat those patients with recurrent symptoms. Decompression is the most commonly performed surgical treatment. [4] It can be done in conjunction with a medial epicondylectomy.

Medial epicondylectomy:[edit | edit source]

An incision is made in a proximal to distal direction parallel to the ulnar nerve. The incision exposes the roof of the nerve and the medial epicondyle. The aponeurotic origin of the flexor mass is then dissected allowing partial excision of the epicondyle. The UCL is not compromised in this procedure and it can be done in addition to the simple decompression. Geutjens et al. found limited evidence to show that the medial anterior epicondylectomy offers a significantly better pain score than the ulnar nerve transposition in the treatment of cubital tunnel syndrome at long-term follow-up.[39]

Anterior transposition (see image[40]):[edit | edit source]

A longitudinal curvilinear incision is made anterior to the medial epicondyle penetrating the roof to expose the nerve. A portion of the medial intermuscular septum is excised which normally provides protection for the ulnar nerve; however, it must be removed in order to expose the nerve for transposition. At the area where the nerve enters the forearm fascia (cubital tunnel), the flexor carpi ulnaris aponeurosis and the deep flexor-pronator aponeurosis are dissected and the nerve is removed from the ulnar groove and moved into the anterior aspect of the arm. Adipose tissue is then sutured from the anterior flap to the medial epicondyle to prevent the nerve from slipping back into the ulnar groove.

Results found no difference in motor nerve conduction velocities or clinical outcome scores, however, between simple decompression and ulnar nerve transposition.[25]

There are three types of anterior transposition techniques in relation to the flexor-pronator mass:

  • Subcutaneous (above) - the goal is to move the ulnar nerve anterior to the elbow axis of flexion, decreasing the tension on the nerve.
  • Intramuscular (within) - proponents of this technique believe that this places the nerve in a straighter line across the elbow joint. Opponents argue that scarring of the nerve can be caused, which serves as the bed for the transposed nerve. The procedure is similar to the subcutaneous trans-positioning, however, a groove is created in the flexor-pronator muscle mass to serve as a tract into which the nerve is transposed.
  • Submuscular (below) - some surgeons prefer to place the nerve completely beneath the flexor-pronator mass. The branches of the medial antebrachial cutaneous nerve are identified and protected. The ulnar nerve is identified and decompressed as in subcutaneous trans-positioning.

Han et al investigated whether fascia wrapping would be a good surgical method and results were positive. Fascia wrapping is a type of sub-fascial trans-positioning. This method provides better immobilisation and requires less dissection than a sub-fascial transposition. . [2]

Selecting a particular surgical approach is based on the etiology of nerve compression, anatomic variations and the surgeon’s experience. With careful protection of the medial antebrachial cutaneous nerve and careful complete decompression of the nerve around the elbow, with or without transposition, good results can be obtained.[1]

Physical Therapy Management[edit | edit source]

Conservative treatment has been shown to have a 90% success rate in acute ulnar irritation with symptoms often resolving within 2-3 months.[41] A nonsurgical approach should be followed for at least a 3 month period before considering a surgical intervention, especially with mild cases.[20] Conservative treatment may include: a 4-6 week period of immobilisation with the elbow splinted at 45 degrees of flexion and the forearm in neutral rotation, activity modification, electrotherapy modalities, anti-inflammatories, soft elbow pads, joint mobilisations, neural flossing [42], neural gliding [43], exercise and patient education.[44][41]

Elbow splint for CBTS.jpg

Active rest is recommended for athletes. Return to throwing is allowed at 4-6 weeks following absence of symptoms with daily activities or exercise and return to full ROM and strength.[26] While there is strong evidence in support of splinting (Figure 2[45]), activity modification and patient education, only low level evidence supports manual techniques such as nerve glides, joint mobilisations and manipulation and exercise.[11][5][45] Despite the low level evidence, improvements have still been seen with manual techniques in patients with cubital tunnel syndrome.

The initial goal of conservative treatment for cubital tunnel syndrome is to control and decrease paraesthesia and pain. When symptoms are mild and aggravating activities can be identified, the first step is to eliminate those pain provoking activities. When symptoms occur in a wider range of activities such as work, therapy becomes more complex and can consist of activity modifications, splinting and rest. With this combination, pain and paraesthesia become more controllable.

Therapy begins with education about the development of symptoms and how certain activities can influence those symptoms. such as stretching or compressing the nerve when collaterally tilting the head or abducting, depressing or externally rotating the shoulder, supinating the forearm or extending the wrist. The patient is educated about pain provoking movements and how best to avoid them in ADLs, which could cause aggravation of the symptoms. For many patients this could mean lifelong management.[5]


Studies show that the effect of rigid night splinting for a period of three months combined with activity modification appears to be successful. Prolonged elbow flexion (static or repetitive) puts strain on the ulnar nerve and increases extraneural and intraneural pressure in the cubital tunnel. The lowest value of these pressures is at an elbow position of 40-50 degrees of flexion. Pressures are significantly higher in full flexion or extension of the elbow. Splinting is designed to alleviate symptoms and prevent the progressive dysfunction of nerves. [26]  Two issues that should be considered however, the ability of the splint to maintain the elbow at the ideal amount of flexion and patient compliance with night splinting.

As well as education, immobilising the elbow with splints can reduce swelling and can help to identify the location of nerve irritation. Splinting the elbow in an appropriate way allows the nerve and the surrounding structures to rest and have relief from traction and compression. This method can be combined with local steroid injections for the relief of pain and swelling. Though steroid injections can have positive effects, caution has be used to avoid complications such as scarring and atrophy.[5]

Avoiding symptom provoking activities can be the most important aspect in treating cubital tunnel syndrome, although this may mean a cessation of work. Creating an effective treatment plan is challenging for professionals as the recovery period of nerves can be unpredictable. Ice application may also help to reduce pain and swelling and can be combined with gently applied active range of motion exercises.

Therapeutic Ultrasound is also an option, but only when used appropriately and with caution as it is also shown to cause further nerve damage when used at an inappropriate intensity, slowing the speed of recovery.

Active range of motion exercises should be initiated within range of comfort, with stretching also within tolerance and only after pain levels have decreased.[5]

Differential diagnosis[edit | edit source]

Differential diagnoses include, but are not limited to:[9][47][5]

  • Cervical Radiculopathy C8-T1: Motor and sensory deficits in a dermatomal pattern including 4th-5th digits, associated weakness of intrinsic muscles of the hand, and associated painful and often limited cervical range of motion.
  • Thoracic Outlet Syndrome (TOS): Compression of the structures of the brachial plexus potentially leading to pain, paresthesias, and weakness in arm, shoulder, and neck.[44]
  • UCL Insufficiency: Laxity of the UCL can lead to excessive or abnormal movement of structures in or around the cubital tunnel creating new sites of compression.
  • Pancoast Tumor: Abnormal growth of tissue on the apex of the lung causing compression of the lower trunk of the brachial plexus.

Early diagnosis is essential for improved recovery, as improvement rates are higher (88%) when treated within one year of onset as opposed to 67% improvement if treated after one year.[9][48]

Additional diagnoses to consider include tophaceous gout and calcium pyrophosphate dehydrate crystal deposition.[26] Cubital tunnel syndrome can often be misdiagnosed as C7 syndrome or other circulatory disturbances, for example Raynaud's disease or polyneuropathy. [4]

Key Research[edit | edit source]

Svernlov et al.[45]
Medium RCT using three groups to compare effectiveness of elbow night splinting, self-nerve glides, and a control. A 3-month period of self-nerve gliding and instruction about the anatomy of the cubital tunnel including information about provocative positions in those with cubital tunnel syndrome resulted in increased pain-free grip strength, decreased daytime pain, and “normalized” EMG studies but was not significantly different from night splinting with instruction or instruction alone. Conclusion: All groups showed statistically significant improvements in COPM scores and night pain compared to baseline, as well as improvements in grip strength, 5th digit adduction power, and EMG studies but these changes did not reflect a statistically significant change.

Zlowodzki et al.[25]
Meta-Analysis of four RCT comparing simple decompression with anterior ulnar nerve transpositions. There were no significant differences between simple decompression and anterior transposition in terms of the clinical scores in those studies (standard mean difference in effect size = -0.04 [95% CI = -0.36 to 0.28], p = 0.81. Authors did not find significant heterogeneity across the studies. Two reports presented post-operative motor nerve conduction velocities; they showed no significant difference between the procedures. Conclusion: Data suggests that simple decompression is a reasonable alternative to anterior transposition for surgical management of ulnar nerve compression at the elbow

Case Reports[edit | edit source]

Coppieters and Bartholomeeusen[11]

The objective was to discuss the diagnosis and treatment of a patient with cubital tunnel syndrome and to illustrate novel treatment modalities for the ulnar nerve and its surrounding structures and target tissues. The patient was a 17 year old female with traumatic onset of cubital tunnel syndrome. She had pain around the elbow and paresthesia in the ulnar nerve distribution. Electrodiagnostic tests were negative.  Segmental cervicothoracic motion dysfunctions were present which were regarded as contributing factors hindering natural recovery. Six treatments included nerve-gliding techniques, segmental joint manipulation, and a home program of nerve gliding and light free-weight exercises. Substantial improvement was recorded on both the impairment and functional level. Symptoms did not recur within 10-month follow-up period. Pain and disability had completely resolved.

Bruce, Wasielewski and Hawke[16]

The patient was a 21-year-old male collegiate wrestler diagnosed with cubital tunnel syndrome. He was diagnosed with cubital tunnel syndrome after 6 weeks of increasing disability and dysfunction. He was treated conservatively for 3 months without resolution of the symptoms. Surgical treatment then involved a subcutaneous ulnar nerve transposition performed to decompress the cubital tunnel. Following surgery, the athlete participated in an aggressive rehabilitation program to restore function and strength to the elbow and adjacent joints. He was cleared for full unrestricted activity 15 days following surgery and returned to athletic competition within one month. Their literature review found no reported cases of cubital tunnel syndrome in wrestlers. Cubital tunnel syndrome is usually seen in throwing athletes and results from either acute trauma or repetitive activities. The athletic trainer should consider cubital tunnel syndrome as a possible pathology for non-throwing athletes when presented with associated signs and symptoms. [16]

Clinical Bottom Line[edit | edit source]

Despite the significant amount of literature devoted to the diagnosis and treatment of cubital tunnel syndrome, optimal treatment often requires two very simple components of treatment - time and rest. While surgical intervention can greatly ease symptoms, successful lifelong management of cubital tunnel syndrome also demands education and a dedicated effort at activity modification. Hopefully, with better environmental work conditions and early detection, can the expenses involved, the time spent in rehabilitation and most importantly the pain and disability patients experience decrease with the help of the patient, employers and the medical management team.[5]

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